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诱导碱中毒对力竭性腿部推举表现的影响。

Effect of induced alkalosis on exhaustive leg press performance.

作者信息

Portington K J, Pascoe D D, Webster M J, Anderson L H, Rutland R R, Gladden L B

机构信息

Department of Health & Human Performance, Auburn University 36849-5323, USA.

出版信息

Med Sci Sports Exerc. 1998 Apr;30(4):523-8. doi: 10.1097/00005768-199804000-00009.

DOI:10.1097/00005768-199804000-00009
PMID:9565933
Abstract

PURPOSE

Fifteen males were studied before, during, and in recovery from exhaustive resistance exercise 105 min after ingesting 0.3 g.kg-1 of either a placebo (white flour) or sodium bicarbonate (NaHCO3).

METHODS

The exercise consisted of five maximal sets on a leg press machine, The load was adjusted to maintain the number of repetitions per set for each subject at approximately 12 repetitions. A significant (P < 0.05) increase in pH (7.40 to 7.47), oxygenated base excess (OxyBE) (-1.3 to 4.0 mEq.L-1), and bicarbonate concentration ([HCO3-]) (22.8 to 27.4 mM) was achieved before exercise with the ingestion of NaHCO3.

RESULTS

The exercise protocol produced significant changes in acid base status consistent with metabolic acidosis for both trials (pH sets 1-5: placebo, 7.4 to 7.26; NaHCO3, 7.47 to 7.33), (OxyBE sets 1-5: placebo, -1.3 to -12.3 mEq.L-1; NaHCO3, 4.0 to -6.9 mEq.L-1) and ([HCO3-] sets 1-5: placebo, 22.9 to 14.0 mM; NaHCO3, 27.4 to 17.6 mM). After every set; pH, OxyBE, and [HCO3-] were significantly higher in the NaHCO3 trial. Blood lactate concentration ([La-]) significantly increased throughout exercise for both trials ([La-] sets 1-5: placebo, 4.6 to 11.3 mM; NaHCO3, 4.8 to 13.4 mM). After sets 4 and 5, blood [La-] was significantly higher in the NaHCO3 trial. Bicarbonate ingestion did not improve performance (total repetitions: NaHCO3 = 59 +/- 3; placebo = 60 +/- 2).

CONCLUSIONS

This may be a result of a lower demand on the whole body metabolic system in comparison with that for other modes of exercise in which ergogenic effects have been found.

摘要

目的

选取15名男性,在摄入0.3 g·kg-1的安慰剂(白面粉)或碳酸氢钠(NaHCO3)后,于力竭性抗阻运动前、运动期间及运动后105分钟恢复阶段进行研究。

方法

运动包括在腿部推举机上进行五组最大重复次数练习,负荷根据每位受试者每组约12次重复的次数进行调整。摄入NaHCO3后,运动前pH值(从7.40升至7.47)、氧合碱剩余(OxyBE,从-1.3升至4.0 mEq·L-1)和碳酸氢盐浓度([HCO3-],从22.8升至27.4 mM)显著升高(P < 0.05)。

结果

两项试验中,运动方案均使酸碱状态发生显著变化,符合代谢性酸中毒(pH值第1 - 5组:安慰剂组,从7.4降至7.26;NaHCO3组,从7.47降至7.33),(OxyBE第1 - 5组:安慰剂组,从-1.3降至-12.3 mEq·L-1;NaHCO3组,从4.0降至-6.9 mEq·L-1)以及([HCO3-]第1 - 5组:安慰剂组,从22.9降至14.0 mM;NaHCO3组,从27.4降至17.6 mM)。每组运动后,NaHCO3试验中的pH值、OxyBE和[HCO3-]均显著更高。两项试验中,运动过程中血乳酸浓度([La-])均显著升高([La-]第1 - 5组:安慰剂组,从4.6升至11.3 mM;NaHCO3组,从4.8升至13.4 mM)。在第4组和第5组运动后,NaHCO3试验中的血[La-]显著更高。摄入碳酸氢盐并未改善运动表现(总重复次数:NaHCO3 = 59 ± 3;安慰剂 = 60 ± 2)。

结论

与已发现具有促力效果的其他运动模式相比,这可能是全身代谢系统需求较低的结果。

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