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1
The use of antibodies to the polypyrimidine tract binding protein (PTB) to analyze the protein components that assemble on alternatively spliced pre-mRNAs that use distant branch points.使用针对多嘧啶序列结合蛋白(PTB)的抗体来分析组装在使用远距离分支点的可变剪接前体mRNA上的蛋白质成分。
RNA. 1998 Jun;4(6):613-25. doi: 10.1017/s1355838298971448.
2
Polypyrimidine tract binding protein interacts with sequences involved in alternative splicing of beta-tropomyosin pre-mRNA.多嘧啶序列结合蛋白与β-原肌球蛋白前体mRNA可变剪接相关序列相互作用。
J Biol Chem. 1992 Dec 15;267(35):25480-7.
3
A neuron-specific splicing switch mediated by an array of pre-mRNA repressor sites: evidence of a regulatory role for the polypyrimidine tract binding protein and a brain-specific PTB counterpart.由一系列前体mRNA阻遏位点介导的神经元特异性剪接开关:多嘧啶序列结合蛋白及脑特异性PTB对应物发挥调控作用的证据
RNA. 1997 Sep;3(9):996-1015.
4
Polypyrimidine tract binding protein functions as a repressor to regulate alternative splicing of alpha-actinin mutally exclusive exons.聚嘧啶序列结合蛋白作为一种阻遏物,调节α-辅肌动蛋白相互排斥外显子的可变剪接。
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Identification of two distinct intron elements involved in alternative splicing of beta-tropomyosin pre-mRNA.鉴定参与β-原肌球蛋白前体mRNA可变剪接的两种不同内含子元件。
Genes Dev. 1990 Jan;4(1):98-110. doi: 10.1101/gad.4.1.98.
6
Alternative splicing of beta-tropomyosin pre-mRNA: multiple cis-elements can contribute to the use of the 5'- and 3'-splice sites of the nonmuscle/smooth muscle exon 6.β-原肌球蛋白前体mRNA的可变剪接:多个顺式元件可影响非肌肉/平滑肌外显子6的5'和3'剪接位点的使用。
Nucleic Acids Res. 1994 Jun 25;22(12):2318-25. doi: 10.1093/nar/22.12.2318.
7
Branch point selection in alternative splicing of tropomyosin pre-mRNAs.原肌球蛋白前体mRNA可变剪接中的分支点选择
Nucleic Acids Res. 1989 Jul 25;17(14):5633-50. doi: 10.1093/nar/17.14.5633.
8
The polypyrimidine tract binding protein (PTB) represses splicing of exon 6B from the beta-tropomyosin pre-mRNA by directly interfering with the binding of the U2AF65 subunit.聚嘧啶序列结合蛋白(PTB)通过直接干扰U2AF65亚基的结合来抑制β-原肌球蛋白前体mRNA中外显子6B的剪接。
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Alternative splicing of beta-tropomyosin pre-mRNA: cis-acting elements and cellular factors that block the use of a skeletal muscle exon in nonmuscle cells.β-原肌球蛋白前体信使核糖核酸的可变剪接:在非肌肉细胞中阻断骨骼肌外显子使用的顺式作用元件和细胞因子。
Genes Dev. 1991 Nov;5(11):2096-107. doi: 10.1101/gad.5.11.2096.
10
The polypyrimidine tract binding protein binds upstream of neural cell-specific c-src exon N1 to repress the splicing of the intron downstream.多嘧啶序列结合蛋白结合在神经细胞特异性c-src基因外显子N1的上游,以抑制下游内含子的剪接。
Mol Cell Biol. 1997 Aug;17(8):4667-76. doi: 10.1128/MCB.17.8.4667.

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Polypyrimidine tract binding protein-1 (PTB1) is a determinant of the tissue and host tropism of a human rhinovirus/poliovirus chimera PV1(RIPO).多嘧啶 tract 结合蛋白-1(PTB1)是决定人鼻病毒/脊髓灰质炎病毒嵌合病毒 PV1(RIPO)组织嗜性和宿主嗜性的决定因素。
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Delivery of bifunctional RNAs that target an intronic repressor and increase SMN levels in an animal model of spinal muscular atrophy.在脊髓性肌萎缩症动物模型中递送靶向内含子阻遏物并提高SMN水平的双功能RNA。
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10
Sam68 regulates a set of alternatively spliced exons during neurogenesis.Sam68在神经发生过程中调控一组可变剪接外显子。
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A neuron-specific splicing switch mediated by an array of pre-mRNA repressor sites: evidence of a regulatory role for the polypyrimidine tract binding protein and a brain-specific PTB counterpart.由一系列前体mRNA阻遏位点介导的神经元特异性剪接开关:多嘧啶序列结合蛋白及脑特异性PTB对应物发挥调控作用的证据
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The polypyrimidine tract binding protein binds upstream of neural cell-specific c-src exon N1 to repress the splicing of the intron downstream.多嘧啶序列结合蛋白结合在神经细胞特异性c-src基因外显子N1的上游,以抑制下游内含子的剪接。
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使用针对多嘧啶序列结合蛋白(PTB)的抗体来分析组装在使用远距离分支点的可变剪接前体mRNA上的蛋白质成分。

The use of antibodies to the polypyrimidine tract binding protein (PTB) to analyze the protein components that assemble on alternatively spliced pre-mRNAs that use distant branch points.

作者信息

Grossman J S, Meyer M I, Wang Y C, Mulligan G J, Kobayashi R, Helfman D M

机构信息

Cold Spring Harbor Laboratory, New York 11724, USA.

出版信息

RNA. 1998 Jun;4(6):613-25. doi: 10.1017/s1355838298971448.

DOI:10.1017/s1355838298971448
PMID:9622121
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1369644/
Abstract

We are using the rat beta-tropomyosin (beta-TM) gene as a model system to study the mechanism of alternative splicing. Previous studies demonstrated that the use of the muscle-specific exon is associated with the use of distant branch points located 147-153 nt upstream of the 3' splice site. In addition, at least one protein, the polypyrimidine tract binding protein (PTB), specifically interacts with critical cis-acting sequences upstream of exon 7 that are involved in blocking the use of this alternative exon in nonmuscle cells. In order to further study the role of PTB, monoclonal antibodies to PTB were prepared. Anti-PTB antibodies did not inhibit the binding of PTB to RNA because they were able to supershift RNA-PTB complexes. To determine if additional proteins interact with sequences within the pre-mRNA, 35S-met-labeled nuclear extracts from HeLa cells were mixed with RNAs and the RNA-protein complexes were recovered by immunoprecipitation using antibodies to PTB. When RNAs containing intron 6 were added to an 35S-met-labeled nuclear extract, precipitation with PTB antibodies showed a novel set of proteins. By contrast, addition of RNAs containing introns 5 or 7 gave the same results as no RNA, indicating that these RNAs are unable to form stable complexes with PTB. These results are in agreement with our previous studies demonstrating that PTB interacts with sequences within intron 6, but not with sequences within introns 5 and 7. When 35S-met-labeled HeLa nuclear extracts were mixed with biotinylated RNA containing intron 6 and the RNA-protein complexes were recovered using streptavidin-agarose beads, an identical pattern of proteins was observed when compared with the immunoprecipitation assay. Analysis of the proteins that assembled on introns 5, 6, or 7 using biotinylated RNA revealed a unique set of proteins that interact with each of these sequences. The composition of proteins interacting with sequences associated with the use of the 3' splice site of intron 6 included proteins of 30, 40, 55, 60, 65, 70, 80, and 100 kDa. Microsequencing identified two of the proteins to be Sam68 and the Far Upstream Element Binding Protein (FBP) from the c-myc gene. In addition, a comparison of the proteins that assemble on introns from the alpha- and beta-TM genes that utilize distant branch points revealed common as well as unique proteins that assemble on these introns. These studies identify a set of proteins, in addition to PTB, that are likely involved in the use of distant branch sites associated with the use of alternatively spliced introns.

摘要

我们正在使用大鼠β-原肌球蛋白(β-TM)基因作为模型系统来研究可变剪接的机制。先前的研究表明,肌肉特异性外显子的使用与位于3'剪接位点上游147 - 153个核苷酸处的远距离分支点的使用相关。此外,至少有一种蛋白质,即多嘧啶序列结合蛋白(PTB),与外显子7上游的关键顺式作用序列特异性相互作用,这些序列参与在非肌肉细胞中阻止该可变外显子的使用。为了进一步研究PTB的作用,制备了针对PTB的单克隆抗体。抗PTB抗体不会抑制PTB与RNA的结合,因为它们能够使RNA - PTB复合物发生超迁移。为了确定是否有其他蛋白质与前体mRNA中的序列相互作用,将来自HeLa细胞的35S - 甲硫氨酸标记的核提取物与RNA混合,并使用针对PTB的抗体通过免疫沉淀回收RNA - 蛋白质复合物。当将含有内含子6的RNA添加到35S - 甲硫氨酸标记的核提取物中时,用PTB抗体沉淀显示出一组新的蛋白质。相比之下,添加含有内含子5或7的RNA得到的结果与不添加RNA时相同,表明这些RNA无法与PTB形成稳定的复合物。这些结果与我们之前的研究一致,即PTB与内含子6中的序列相互作用,但不与内含子5和7中的序列相互作用。当将35S - 甲硫氨酸标记的HeLa核提取物与含有内含子6的生物素化RNA混合,并使用链霉亲和素 - 琼脂糖珠回收RNA - 蛋白质复合物时,与免疫沉淀分析相比,观察到相同的蛋白质模式。使用生物素化RNA对在内含子5、6或7上组装的蛋白质进行分析,揭示了一组与这些序列中的每一个相互作用的独特蛋白质。与内含子6的3'剪接位点使用相关序列相互作用的蛋白质组成包括3 kDa、40 kDa、55 kDa、60 kDa、65 kDa、70 kDa、80 kDa和100 kDa的蛋白质。微量测序确定其中两种蛋白质为Sam68和来自c - myc基因的远上游元件结合蛋白(FBP)。此外,对利用远距离分支点的α - 和β - TM基因内含子上组装的蛋白质进行比较,发现了在这些内含子上组装的共同以及独特的蛋白质。这些研究确定了一组除PTB之外的蛋白质,它们可能参与与可变剪接内含子使用相关的远距离分支位点的使用。