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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Thurston H, Swales J D

Clin Sci Mol Med. 1976 Sep;51(3):275-9. doi: 10.1042/cs0510275.

Restriction of dietary salt intake does not affect the development or maintenance of hypertension in rats with unilateral renal ischaemia whether the contralateral kidney is present (Goldblatt 2 model) or not (Goldblatt 1 model). 2. Acute dietary salt depletion induces a similar loss of sodium and fall in body weight with little change in blood pressure in both normal and hypertensive rats. 3. Excision of the ischaemic kidney in rats with short-term (less than 50 days) Goldblatt 2 hypertension restores the mean blood pressure to normal, whereas Goldblatt 1 hypertensive rats show only a partial response. Previous salt depletion of this model enhances the blood pressure response to nephrectomy. 4. Sodium retention plays no part in the development or maintenance of Goldblatt 2 kidney hypertension. However, although sodium retention is normally involved in the Goldblatt 1 model, hypertension can develop in the absence of dietary sodium.

限制饮食中的盐摄入量对单侧肾缺血大鼠高血压的发生或维持没有影响，无论对侧肾脏是否存在（戈德布拉特2型模型）。2. 急性饮食性盐缺乏在正常大鼠和高血压大鼠中都会导致类似的钠流失和体重下降，而血压变化很小。3. 短期（少于50天）戈德布拉特2型高血压大鼠切除缺血肾后，平均血压恢复正常，而戈德布拉特1型高血压大鼠仅表现出部分反应。该模型先前的盐缺乏会增强肾切除术后的血压反应。4. 钠潴留在戈德布拉特2型肾性高血压的发生或维持中不起作用。然而，尽管钠潴留通常与戈德布拉特1型模型有关，但在没有饮食钠的情况下也会发生高血压。

Thurston H, Swales J D

Clin Sci Mol Med. 1976 Sep;51(3):275-9. doi: 10.1042/cs0510275.

Restriction of dietary salt intake does not affect the development or maintenance of hypertension in rats with unilateral renal ischaemia whether the contralateral kidney is present (Goldblatt 2 model) or not (Goldblatt 1 model). 2. Acute dietary salt depletion induces a similar loss of sodium and fall in body weight with little change in blood pressure in both normal and hypertensive rats. 3. Excision of the ischaemic kidney in rats with short-term (less than 50 days) Goldblatt 2 hypertension restores the mean blood pressure to normal, whereas Goldblatt 1 hypertensive rats show only a partial response. Previous salt depletion of this model enhances the blood pressure response to nephrectomy. 4. Sodium retention plays no part in the development or maintenance of Goldblatt 2 kidney hypertension. However, although sodium retention is normally involved in the Goldblatt 1 model, hypertension can develop in the absence of dietary sodium.

限制饮食中的盐摄入量对单侧肾缺血大鼠高血压的发生或维持没有影响，无论对侧肾脏是否存在（戈德布拉特2型模型）。2. 急性饮食性盐缺乏在正常大鼠和高血压大鼠中都会导致类似的钠流失和体重下降，而血压变化很小。3. 短期（少于50天）戈德布拉特2型高血压大鼠切除缺血肾后，平均血压恢复正常，而戈德布拉特1型高血压大鼠仅表现出部分反应。该模型先前的盐缺乏会增强肾切除术后的血压反应。4. 钠潴留在戈德布拉特2型肾性高血压的发生或维持中不起作用。然而，尽管钠潴留通常与戈德布拉特1型模型有关，但在没有饮食钠的情况下也会发生高血压。