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Tissue injury in neutrophilic inflammation.

作者信息

Ginsburg I

出版信息

Inflamm Res. 1998 Jun;47(6):237-8. doi: 10.1007/s000110050323.

DOI:10.1007/s000110050323
PMID:9683029
Abstract
摘要

相似文献

1
Tissue injury in neutrophilic inflammation.
Inflamm Res. 1998 Jun;47(6):237-8. doi: 10.1007/s000110050323.
2
Neutrophilic leukocyte granules: from structure to function.中性粒细胞颗粒:从结构到功能
Adv Exp Med Biol. 1993;336:17-33. doi: 10.1007/978-1-4757-9182-2_3.
3
Fine structure of the camel neutrophilic granulocytes with special references to its function.骆驼嗜中性粒细胞的精细结构及其功能的特殊参考
Z Mikrosk Anat Forsch. 1989;103(6):985-92.
4
[Blood neutrophilic leukocytes in patients with suppurative-septic diseases].[化脓性-败血性疾病患者的血液中性粒细胞]
Arkh Patol. 1994 Sep-Oct;56(5):20-5.
5
Neutrophil-mediated vascular injury.中性粒细胞介导的血管损伤。
Acta Med Scand Suppl. 1987;715:123-9. doi: 10.1111/j.0954-6820.1987.tb09912.x.
6
Electron microscopic findings of parallel tubular granules in a case of chronic neutrophilic leukaemia.一例慢性中性粒细胞白血病中平行管状颗粒的电子显微镜检查结果
J Clin Pathol. 1994 Apr;47(4):367-9. doi: 10.1136/jcp.47.4.367.
7
Tissue injury in neutrophilic inflammation.嗜中性粒细胞炎症中的组织损伤。
Inflamm Res. 1997 Oct;46(10):382-91. doi: 10.1007/s000110050208.
8
Neutrophil specific granules: a fuse that ignites the inflammatory response.中性粒细胞特异性颗粒:引发炎症反应的导火索。
Clin Res. 1984 Sep;32(3):320-8.
9
Granules and vesicles of human neutrophils. The role of endomembranes as source of plasma membrane proteins.人类中性粒细胞的颗粒与囊泡。内膜作为质膜蛋白来源的作用。
Eur J Haematol. 1993 Nov;51(5):318-22. doi: 10.1111/j.1600-0609.1993.tb01615.x.
10
[Ultrastructural characteristics of the peripheral blood neutrophilic leukocytes and the quantitative assessment of the cytoplasmic granules in eczema patients].[湿疹患者外周血中性粒细胞的超微结构特征及细胞质颗粒的定量评估]
Vestn Dermatol Venerol. 1986(1):11-4.

本文引用的文献

1
Diethyldithiocarbamate and nitric oxide synergize with oxidants and with membrane-damaging agents to injure mammalian cells.二乙基二硫代氨基甲酸盐和一氧化氮与氧化剂及膜损伤剂协同作用损伤哺乳动物细胞。
Free Radic Res. 1997 Aug;27(2):143-64. doi: 10.3109/10715769709097847.
2
Exogenous fatty acids modulate the functional and cytotoxic responses of cultured pulmonary artery endothelial cells to oxidant stress.外源性脂肪酸可调节培养的肺动脉内皮细胞对氧化应激的功能和细胞毒性反应。
J Lab Clin Med. 1997 May;129(5):548-56. doi: 10.1016/s0022-2143(97)90009-3.
3
Cytosolic phospholipase A2 (PLA2), but not secretory PLA2, potentiates hydrogen peroxide cytotoxicity in kidney epithelial cells.
J Biol Chem. 1996 Aug 30;271(35):21505-13. doi: 10.1074/jbc.271.35.21505.
4
H2O2 renders cells accessible to lysis by exogenous phospholipase A2: a novel mechanism for cell damage in inflammatory processes.过氧化氢使细胞在外源磷脂酶A2作用下易于裂解:炎症过程中细胞损伤的一种新机制。
FEBS Lett. 1996 Mar 25;383(1-2):75-8. doi: 10.1016/0014-5793(96)00227-x.
5
Neutrophil oxygen radical generation. Synergistic responses to tumor necrosis factor and mono/polyunsaturated fatty acids.中性粒细胞氧自由基生成。对肿瘤坏死因子和单/多不饱和脂肪酸的协同反应。
J Clin Invest. 1996 Apr 1;97(7):1605-9. doi: 10.1172/JCI118585.
6
Killing of endothelial cells and release of arachidonic acid. Synergistic effects among hydrogen peroxide, membrane-damaging agents, cationic substances, and proteinases and their modulation by inhibitors.内皮细胞的杀伤及花生四烯酸的释放。过氧化氢、膜损伤剂、阳离子物质和蛋白酶之间的协同作用及其受抑制剂的调节。
Inflammation. 1993 Jun;17(3):295-319. doi: 10.1007/BF00918992.
7
Ethanol synergizes with hydrogen peroxide, peroxyl radical, and trypsin to kill epithelial cells in culture.乙醇与过氧化氢、过氧自由基和胰蛋白酶协同作用,以杀死培养中的上皮细胞。
Free Radic Biol Med. 1994 Feb;16(2):263-9. doi: 10.1016/0891-5849(94)90151-1.
8
Can hemolytic streptococci be considered "forefathers" of modern phagocytes? Both cell types freely migrate in tissues and destroy host cells by a "synergistic cross-talk" among their secreted agonists.溶血性链球菌能被视为现代吞噬细胞的“鼻祖”吗?这两种细胞类型都能在组织中自由迁移,并通过其分泌的激动剂之间的“协同串扰”来破坏宿主细胞。
Comp Biochem Physiol C Pharmacol Toxicol Endocrinol. 1994 Oct;109(2):147-58.
9
Synergistic effects among oxidants, membrane-damaging agents, fatty acids, proteinases, and xenobiotics: killing of epithelial cells and release of arachidonic acid.氧化剂、膜损伤剂、脂肪酸、蛋白酶和外源性物质之间的协同作用:上皮细胞的杀伤及花生四烯酸的释放。
Inflammation. 1995 Feb;19(1):101-18. doi: 10.1007/BF01534384.
10
Cell damage in inflammatory and infectious sites might involve a coordinated "cross-talk" among oxidants, microbial haemolysins and ampiphiles, cationic proteins, phospholipases, fatty acids, proteinases and cytokines (an overview).炎症和感染部位的细胞损伤可能涉及氧化剂、微生物溶血素和两亲分子、阳离子蛋白、磷脂酶、脂肪酸、蛋白酶和细胞因子之间协调的“相互作用”(概述)。
Free Radic Res. 1995 Jun;22(6):489-517. doi: 10.3109/10715769509150323.