出生时用干扰素制剂治疗的小鼠中的进行性肾小球肾炎。 - Suppr

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超能文献

Progressive glomerulonephritis in mice treated with interferon preparations at birth.

作者信息

Gresser I, Maury C, Tovey M, Morel-Maroger L, Pontillon F

出版信息

Nature. 1976 Sep 30;263(5576):420-2. doi: 10.1038/263420a0.

DOI:10.1038/263420a0

PMID:972680

Abstract

摘要

相似文献

Progressive glomerulonephritis in mice treated with interferon preparations at birth.出生时用干扰素制剂治疗的小鼠中的进行性肾小球肾炎。

Nature. 1976 Sep 30;263(5576):420-2. doi: 10.1038/263420a0.

An ultrastructural study of the development of nephritis in mice treated with interferon in the neonatal period.

Lab Invest. 1978 Nov;39(5):513-22.

Severity of glomerulonephritis induced in different strains of suckling mice by infection with lymphocytic choriomeningitis virus: correlation with amounts of endogenous interferon and circulating immune complexes.淋巴细胞性脉络丛脑膜炎病毒感染诱导不同品系乳鼠发生肾小球肾炎的严重程度：与内源性干扰素量及循环免疫复合物的相关性

J Pathol. 1982 Dec;138(4):325-36. doi: 10.1002/path.1711380404.

Role of interferon in the genesis of experimental glomerulonephritis in mice.干扰素在小鼠实验性肾小球肾炎发病机制中的作用。

Contrib Microbiol Immunol. 1983;7:73-82.

[C-type murine leukemia virus and the pathogenesis of necrotizing arteritis and glomerulonephritis in MRL/1 mice].[C型鼠白血病病毒与MRL/1小鼠坏死性动脉炎和肾小球肾炎的发病机制]

Hokkaido Igaku Zasshi. 1982 Nov;57(6):688-95.

Case of glomerulonephritis associated with acute toxoplasmosis.

Br Med J. 1974 Sep 14;3(5932):664-5. doi: 10.1136/bmj.3.5932.664.

[Glomerulonephritis caused by a shunt: description of a case].

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Serial biopsies in diffuse proliferative glomerulonephritis in adults: an attempt for a better understanding of sporadic acute glomerulonephritis.成人弥漫性增生性肾小球肾炎的系列活检：旨在更好地理解散发性急性肾小球肾炎的尝试

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Anti-interferon globulin inhibits the development of glomerulonephritis in mice infected at birth with lymphocytic choriomeningitis virus.抗干扰素球蛋白可抑制出生时感染淋巴细胞性脉络丛脑膜炎病毒的小鼠肾小球肾炎的发展。

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Experimental infections with African Trypanosomes. VI. Glomerulonephritis involving the alternate pathway of complement activation.非洲锥虫的实验性感染。VI. 涉及补体激活替代途径的肾小球肾炎。

Am J Trop Med Hyg. 1974 Jan;23(1):15-26.

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Neutralization of interferon-gamma in neonatal SOCS1-/- mice prevents fatty degeneration of the liver but not subsequent fatal inflammatory disease.中和新生期SOCS1基因敲除小鼠体内的干扰素-γ可预防肝脏脂肪变性，但无法预防随后出现的致命性炎症疾病。

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