Marié R M, Rossa Y, Lambert J, Vérard L, Marchal G, Viader F
Service de Neurologie Vastel, CHU de Caen.
Rev Neurol (Paris). 1998 Jan;154(1):28-34.
A 63 year-old-man, with an opercular infarct had a motor deficit with ataxic hemiparesis and cerebellar dysarthria. The motor deficit disappeared, but the cerebellar signs remained after three months. A voice instrumental analysis confirmed the cerebellar pattern of the dysarthria. CT and MRI showed the opercular lesion and the normality of the cerebellum and the pons. A volumic MRI analysis precised the localisation and the volume of the lesion. The oxygen consumption (CMRO2) was measured with PET and showed no cerebellar diaschisis. We propose that, in this case, the cerebellar dysarthria is the orofacial ataxic component of the hemiparesis. We suggest that it is related to disruption of the cortico-cerebellar tract, connecting the right orofacial frontal area of the primary motor cortex with the paravermal segment of the left cerebellar hemisphere.
一名63岁男性,患有岛盖部梗死,出现运动功能障碍,表现为共济失调性偏瘫和小脑性构音障碍。运动功能障碍消失,但三个月后小脑体征仍存在。语音仪器分析证实了构音障碍的小脑模式。CT和MRI显示岛盖部病变,小脑和脑桥正常。容积性MRI分析明确了病变的定位和体积。通过PET测量氧耗量(CMRO2),未显示小脑远隔性机能障碍。我们提出,在这种情况下,小脑性构音障碍是偏瘫的口面部共济失调成分。我们认为它与连接初级运动皮层右侧口面部额叶区域和左小脑半球旁中央小叶的皮质-小脑束中断有关。