相似文献
1
Cooperation between the Cdk inhibitors p27(KIP1) and p57(KIP2) in the control of tissue growth and development.
Genes Dev. 1998 Oct 15;12(20):3162-7. doi: 10.1101/gad.12.20.3162.
2
Disappearance of p27(KIP1) and increase in proliferation of the lens cells after extraction of most of the fiber cells of the lens.
Curr Eye Res. 2005 Jun;30(6):437-42. doi: 10.1080/02713680590959286.
3
Persistent and heterogenous expression of the cyclin-dependent kinase inhibitor, p27KIP1, in rat hearts during development.
J Mol Cell Cardiol. 1998 Mar;30(3):463-74. doi: 10.1006/jmcc.1997.0611.
4
p27Kip1 and p57Kip2 regulate proliferation in distinct retinal progenitor cell populations.
J Neurosci. 2001 Jun 15;21(12):4259-71. doi: 10.1523/JNEUROSCI.21-12-04259.2001.
5
Suppression of cell transformation by the cyclin-dependent kinase inhibitor p57KIP2 requires binding to proliferating cell nuclear antigen.
Proc Natl Acad Sci U S A. 1998 Feb 17;95(4):1392-7. doi: 10.1073/pnas.95.4.1392.
6
Cloning of p57KIP2, a cyclin-dependent kinase inhibitor with unique domain structure and tissue distribution.
Genes Dev. 1995 Mar 15;9(6):639-49. doi: 10.1101/gad.9.6.639.
7
Ablation of the CDK inhibitor p57Kip2 results in increased apoptosis and delayed differentiation during mouse development.
Genes Dev. 1997 Apr 15;11(8):973-83. doi: 10.1101/gad.11.8.973.
8
The cyclin-dependent kinase inhibitor p57Kip2 regulates cell cycle exit, differentiation, and migration of embryonic cerebral cortical precursors.
Cereb Cortex. 2011 Aug;21(8):1840-56. doi: 10.1093/cercor/bhq254. Epub 2011 Jan 18.
9
Involvement of p27KIP1 in the proliferation of the developing corneal endothelium.
Invest Ophthalmol Vis Sci. 2004 Jul;45(7):2163-7. doi: 10.1167/iovs.03-1238.
10
Distinct developmental roles of cell cycle inhibitors p57Kip2 and p27Kip1 distinguish pituitary progenitor cell cycle exit from cell cycle reentry of differentiated cells.
Mol Cell Biol. 2009 Apr;29(7):1895-908. doi: 10.1128/MCB.01885-08. Epub 2009 Jan 12.
引用本文的文献
1
Mettl3 Regulates Lens Development by Promoting the Differentiation Processes of Secondary Fiber Cells.
Invest Ophthalmol Vis Sci. 2025 Jul 1;66(9):45. doi: 10.1167/iovs.66.9.45.
2
Transcriptome Meta-Analysis Uncovers Cell-Specific Regulatory Relationships in Embryonic, Juvenile, Adult, and Aged Mouse Lens Epithelium and Fibers.
Invest Ophthalmol Vis Sci. 2025 Apr 1;66(4):42. doi: 10.1167/iovs.66.4.42.
3
Regenerative therapies for myocardial infarction: exploring the critical role of energy metabolism in achieving cardiac repair.
Front Cardiovasc Med. 2025 Feb 7;12:1533105. doi: 10.3389/fcvm.2025.1533105. eCollection 2025.
4
JAM-C Is Important for Lens Epithelial Cell Proliferation and Lens Fiber Maturation in Murine Lens Development.
Invest Ophthalmol Vis Sci. 2023 Dec 1;64(15):15. doi: 10.1167/iovs.64.15.15.
5
Uterine-specific deletion enhances stromal cell senescence and impairs placentation, resulting in pregnancy loss.
iScience. 2023 Jun 8;26(7):107028. doi: 10.1016/j.isci.2023.107028. eCollection 2023 Jul 21.
6
FOXG1 targets BMP repressors and cell cycle inhibitors in human neural progenitor cells.
Hum Mol Genet. 2023 Jul 20;32(15):2511-2522. doi: 10.1093/hmg/ddad089.
7
Multiomics Analysis Reveals Novel Genetic Determinants for Lens Differentiation, Structure, and Transparency.
Biomolecules. 2023 Apr 19;13(4):693. doi: 10.3390/biom13040693.
8
Repurposing a Cyclin-Dependent Kinase 1 (CDK1) Mitotic Regulatory Network to Complete Terminal Differentiation in Lens Fiber Cells.
Invest Ophthalmol Vis Sci. 2023 Feb 1;64(2):6. doi: 10.1167/iovs.64.2.6.
9
Generation of Lens Progenitor Cells and Lentoid Bodies from Pluripotent Stem Cells: Novel Tools for Human Lens Development and Ocular Disease Etiology.
Cells. 2022 Nov 6;11(21):3516. doi: 10.3390/cells11213516.
10
Deficiency of the bZIP transcription factors Mafg and Mafk causes misexpression of genes in distinct pathways and results in lens embryonic developmental defects.
Front Cell Dev Biol. 2022 Aug 26;10:981893. doi: 10.3389/fcell.2022.981893. eCollection 2022.
本文引用的文献
1
The retinoblastoma gene family: cousins with overlapping interests.
Trends Genet. 1998 Jun;14(6):223-9. doi: 10.1016/s0168-9525(98)01470-x.
2
Rb binds c-Jun and activates transcription.
EMBO J. 1998 Apr 15;17(8):2342-52. doi: 10.1093/emboj/17.8.2342.
3
Sox1 directly regulates the gamma-crystallin genes and is essential for lens development in mice.
Genes Dev. 1998 Mar 15;12(6):776-81. doi: 10.1101/gad.12.6.776.
4
Stable binding to E2F is not required for the retinoblastoma protein to activate transcription, promote differentiation, and suppress tumor cell growth.
Genes Dev. 1998 Jan 1;12(1):95-106. doi: 10.1101/gad.12.1.95.
5
Distinct roles for E2F proteins in cell growth control and apoptosis.
Proc Natl Acad Sci U S A. 1997 Jul 8;94(14):7245-50. doi: 10.1073/pnas.94.14.7245.
6
Altered cell differentiation and proliferation in mice lacking p57KIP2 indicates a role in Beckwith-Wiedemann syndrome.
Nature. 1997 May 8;387(6629):151-8. doi: 10.1038/387151a0.
7
Genetic variation among 129 substrains and its importance for targeted mutagenesis in mice.
Nat Genet. 1997 May;16(1):19-27. doi: 10.1038/ng0597-19.
8
Ablation of the CDK inhibitor p57Kip2 results in increased apoptosis and delayed differentiation during mouse development.
Genes Dev. 1997 Apr 15;11(8):973-83. doi: 10.1101/gad.11.8.973.
9
Retinoblastoma protein in growth suppression and death protection.
Curr Opin Genet Dev. 1997 Feb;7(1):39-45. doi: 10.1016/s0959-437x(97)80107-4.
10
Rb functions to inhibit apoptosis during myocyte differentiation.
Cancer Res. 1997 Feb 1;57(3):351-4.
Zotero 插件