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[慢性心力衰竭的体育锻炼康复治疗]

[Rehabilitation by physical exercise in chronic heart failure].

作者信息

Broustet J P, Douard H, Parrens E, Labbé L

机构信息

Service de cardiologie A, Hôpital cardiologique Haut-Lévêque, Pessac, France.

出版信息

Arch Mal Coeur Vaiss. 1998 Nov;91(11):1399-405.

PMID:9864610
Abstract

The aim of rehabilitation is to improve exercise capacity and, thereby, the autonomy of patients with cardiac failure. For many years, these patients were considered inapt to perform physical exercise and they are in the same situation at the dawn of the year 2000 as patients with myocardial infarction forty years ago. The symptoms of cardiac failure (dyspnoea of effort and muscular fatigue) are not only the consequence of pulmonary hypertension and decreased muscular perfusion. Prolonged interruption of exercise and long stays in bed or in a chair lead to anatomical and functional amyotrophy, which, in turns, incites to further inactivity. Deconditioned respiratory muscles cannot tolerate the increased load of hyperventilation. Neurohormonal changes cause vasoconstriction which reduces muscular perfusion. Physical training can significantly improve these abnormalities, though it does not seem to have a measurable effect on cardiac function; based on segmental work which enables performance of substantial efforts with a minimum of haemodynamic changes, it provides a 20 to 30% gain in capacity, mainly increasing the duration of submaximal exercise rather than maximum performance. Muscular fatigue is the symptom which is the most improved. Unfortunately the organisation, which is more difficult than in the post-infarction period, and the generalisation of the practice of long-term, well adapted physical training remains marginal although hundreds of thousands of patients could benefit; more than the inertia of the official instances concerning anything related to cardiac rehabilitation, it is the lack of interest shown by cardiologists and the absence of flexible structures within the health care organisation for elderly people which are responsible.

摘要

康复的目的是提高运动能力,从而改善心力衰竭患者的自主性。多年来,这些患者被认为不适合进行体育锻炼,在2000年初,他们的情况与40年前的心肌梗死患者相同。心力衰竭的症状(劳力性呼吸困难和肌肉疲劳)不仅是肺动脉高压和肌肉灌注减少的结果。运动的长期中断以及长时间卧床或坐在椅子上会导致解剖学和功能性肌萎缩,进而促使进一步的不活动。呼吸肌失健不能耐受过度通气增加的负荷。神经激素变化导致血管收缩,从而减少肌肉灌注。体育锻炼可以显著改善这些异常情况,尽管它似乎对心脏功能没有可测量的影响;基于能够以最小的血流动力学变化进行大量运动的分段训练,它能使运动能力提高20%至30%,主要是增加次最大运动的持续时间而非最大运动表现。肌肉疲劳是改善最明显的症状。不幸的是,尽管成千上万的患者可能受益,但康复的组织工作比心肌梗死后时期更困难,长期、适应性良好的体育锻炼的普及仍然有限;与官方机构在心脏康复相关任何事情上的惰性相比,更主要的是心脏病专家缺乏兴趣以及医疗保健机构中缺乏针对老年人的灵活结构。

相似文献

1
[Rehabilitation by physical exercise in chronic heart failure].[慢性心力衰竭的体育锻炼康复治疗]
Arch Mal Coeur Vaiss. 1998 Nov;91(11):1399-405.
2
Feasibility of physical training after myocardial infarction and its effect on return to work, morbidity and mortality.心肌梗死后体育锻炼的可行性及其对重返工作、发病率和死亡率的影响。
Acta Med Scand Suppl. 1976;599:7-84.
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Przegl Lek. 2004;61(2):105-8.
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[Segmental rehabilitation in cardiac failure: short and long-term results].[心力衰竭的节段性康复:短期和长期结果]
Arch Mal Coeur Vaiss. 1996 Jul;89(7):819-24.
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6
Effect of physical training on exercise capacity, gas exchange and N-terminal pro-brain natriuretic peptide levels in patients with chronic heart failure.体育锻炼对慢性心力衰竭患者运动能力、气体交换及N末端脑钠肽前体水平的影响
Eur J Cardiovasc Prev Rehabil. 2006 Oct;13(5):812-7. doi: 10.1097/01.hjr.0000238396.42718.61.
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The time course of haemodynamic, autonomic and skeletal muscle metabolic abnormalities following first extensive myocardial infarction in man.人类首次大面积心肌梗死后血流动力学、自主神经及骨骼肌代谢异常的时间进程。
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[Physical exercise in older patients with chronic heart failure].
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Medicina (Kaunas). 2024 Sep 26;60(10):1583. doi: 10.3390/medicina60101583.