甲基汞中毒、临床随访及感觉神经传导研究
Methylmercury poisoning, Clinical follow-up and sensory nerve conduction studies.
作者信息
Snyder R D, Seelinger D F
出版信息
J Neurol Neurosurg Psychiatry. 1976 Jul;39(7):701-4. doi: 10.1136/jnnp.39.7.701.
Abstract
Methylmercury poisoning occurred in four cases after passage of methylmercury through the food chain. The neurological damage in all four cases was severe. The damage was greater at younger ages with maximum involvement in the case of transplacental poisoning. Significant recovery occurred in two cases, but on six-year follow-up two cases remained severely impaired. Clinical and electrophysiological evidence suggests that damage to peripheral sensory nerves may not be the cause of the late sensory symptomatology.
摘要
甲基汞通过食物链传递后，有4例发生了甲基汞中毒。所有4例的神经损伤都很严重。年龄越小损伤越严重，经胎盘中毒的病例受累程度最大。2例有明显恢复，但在6年随访时，仍有2例严重受损。临床和电生理证据表明，外周感觉神经损伤可能不是晚期感觉症状的原因。
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