草酸中毒。儿童期骨髓痨的一种罕见病因。
Oxalosis. An unusual cause of myelophthisis in childhood.
作者信息
McKenna R W, Dehner L P
出版信息
Am J Clin Pathol. 1976 Dec;66(6):991-7. doi: 10.1093/ajcp/66.6.991.
PMID:998570
Abstract
In a child with renal failure and oliguria due to hyperoxaluria myelophthisis developed as a result of extensive bone-marrow replacement with calcium oxalate crystals and an accompanying fibrous proliferations. The histopathology associated with this metabolic disorder was demonstrated in posterior iliac crest bone-marrow trephine biopsies, renal biopsies, and nephrectomy specimens. Crystals were demonstrated in biopsy specimens of transplanted kidneys within six weeks following renal transplantation.
摘要
在一名因高草酸尿症导致肾衰竭和少尿的儿童中，骨髓痨是由于草酸钙晶体广泛替代骨髓以及伴随的纤维组织增生而发展起来的。这种代谢紊乱相关的组织病理学在髂后嵴骨髓环钻活检、肾活检和肾切除标本中得到证实。在肾移植后六周内，移植肾的活检标本中发现了晶体。
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引用本文的文献
1
[Recurrence of the original disease in the transplanted kidney].
Klin Wochenschr. 1984 Apr 2;62(7):289-98. doi: 10.1007/BF01716444.
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Bone disease of primary hyperoxaluria in infancy.
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A contribution to the formation mechanism of calcium oxalate urinary calculi. IV. Experimental investigations of the intrarenal crystallisation of calcium oxalate in rabbit.
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