James D V, Doust J H
Cheltenham and Gloucester College of Higher Education, Leisure and Sport Research Unit, Gloucestershire, UK.
Eur J Appl Physiol Occup Physiol. 1999 Feb;79(3):237-43. doi: 10.1007/s004210050501.
Elevated oxygen uptake (VO2) during moderate-intensity running following a bout of interval running training has been studied previously. To further investigate this phenomenon, the VO2 response to high-intensity exercise was examined following a bout of interval running. Well-trained endurance runners were split into an experimental group [maximum oxygen uptake, VO2max 4.73 (0.39)l x min(-1)] and a reliability group [VO2max 4.77 (0.26)l x min(-1)]. The experimental group completed a training session (4 x 800 m at 1 km x h(-1) below speed at VO2max, with 3 min rest between each 800-m interval). Five minutes prior to, and 1 h following the training session, subjects completed 6 min 30 s of constant speed, high-intensity running designed to elicit 40% delta (where delta is the difference between VO2 at ventilatory threshold and VO2max; tests 1 and 2, respectively). The slow component of VO2 kinetics was quantified as the difference between the VO2 at 6 min and the VO2 at 3 min of exercise, i.e. deltaVO2(6-3). The deltaVO2(-3) was the same in two identical conditions in the reliability group [mean (SD): 0.30 (0.10)l x min(-1) vs 0.32 (0.13)l x min(-1)]. In the experimental group, the magnitude of the slow component of VO2 kinetics was increased in test 2 compared with test 1 by 24.9% [0.27 (0.14)l x min(-1) vs 0.34 (0.08)l x min(-1), P < 0.05]. The increase in deltaVO2(6-3) in the experimental group was observed in the absence of any significant change in body mass, core temperature or blood lactate concentration, either at the start or end of tests 1 or 2. It is concluded that similar mechanisms may be responsible for the slow component of VO2 kinetics and for the fatigue following the training session. It has been suggested previously that this mechanism may be linked primarily to changes within the active limb, with the recruitment of alternative and/or additional less efficient fibres.
之前已经对间歇跑训练后中等强度跑步时摄氧量（VO₂）升高的情况进行过研究。为了进一步探究这一现象，在间歇跑训练后，对VO₂对高强度运动的反应进行了检测。训练有素的耐力跑者被分为实验组[最大摄氧量，VO₂max 4.73（0.39）升·分钟⁻¹]和可靠性对照组[VO₂max 4.77（0.26）升·分钟⁻¹]。实验组完成了一次训练课（以比VO₂max时的速度慢1千米·小时⁻¹的速度进行4组800米跑，每组800米间隔之间休息3分钟）。在训练课开始前5分钟以及结束后1小时，受试者完成了6分30秒的恒定速度高强度跑步，旨在诱发40%的差值（差值是通气阈值时的VO₂与VO₂max之间的差值；分别为测试1和测试2）。VO₂动力学的慢成分被量化为运动6分钟时的VO₂与运动3分钟时的VO₂之间的差值，即ΔVO₂(6 - 3)。在可靠性对照组的两个相同条件下，ΔVO₂(-3)相同[均值（标准差）：0.30（0.10）升·分钟⁻¹对0.32（0.13）升·分钟⁻¹]。在实验组中，与测试1相比，测试2中VO₂动力学慢成分的幅度增加了24.9%[0.27（0.14）升·分钟⁻¹对0.34（0.08）升·分钟⁻¹，P < 0.05]。在测试1或测试2开始或结束时，实验组中ΔVO₂(6 - 3)的增加是在体重、核心温度或血乳酸浓度没有任何显著变化的情况下观察到的。得出的结论是，类似的机制可能是VO₂动力学慢成分以及训练课后疲劳的原因。之前有人提出，这种机制可能主要与活动肢体内部的变化有关，涉及替代和/或额外的效率较低的纤维的募集。
