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From lesions to leptin: hypothalamic control of food intake and body weight.

作者信息

Elmquist J K, Elias C F, Saper C B

机构信息

Department of Neurology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts 02215, USA.

出版信息

Neuron. 1999 Feb;22(2):221-32. doi: 10.1016/s0896-6273(00)81084-3.

DOI:10.1016/s0896-6273(00)81084-3
PMID:10069329
Abstract
摘要

相似文献

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From lesions to leptin: hypothalamic control of food intake and body weight.从病变到瘦素:下丘脑对食物摄入和体重的控制
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Hypothalamic control of feeding.下丘脑对进食的控制。
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Role of hypothalamic melanocortin 3/4-receptors in mediating chronic cardiovascular, renal, and metabolic actions of leptin.下丘脑黑皮质素3/4受体在介导瘦素的慢性心血管、肾脏及代谢作用中的作用。
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Leptin-induced leptin resistant rats exhibit enhanced responses to the melanocortin agonist MT II.瘦素诱导的瘦素抵抗大鼠对促黑素激动剂MT II表现出增强的反应。
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Identification of nesfatin-1 as a satiety molecule in the hypothalamus.在下丘脑将nesfatin-1鉴定为一种饱腹感分子。
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Melanocortin and leptin signaling systems: central regulation of catabolic energy balance.
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Evidence that dark-phase hyperphagia induced by neurotoxin 6-hydroxydopamine may be due to decreased leptin and increased neuropeptide Y signaling.有证据表明,神经毒素6-羟基多巴胺诱导的暗相多食可能是由于瘦素减少和神经肽Y信号增加所致。
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Effects of vagal and splanchnic section on food intake, weight, serum leptin and hypothalamic neuropeptide Y in rat.迷走神经和内脏神经切断对大鼠食物摄入量、体重、血清瘦素和下丘脑神经肽Y的影响。
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Neuropeptides responding to leptin.
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