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First description in Europe of the emergence of Enterococcus faecium ST117 carrying both vanA and vanB genes, isolated in Greece.首次在欧洲描述携带 vanA 和 vanB 基因的屎肠球菌 ST117 的出现，该菌在希腊被分离。
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The VanS sensor negatively controls VanR-mediated transcriptional activation of glycopeptide resistance genes of Tn1546 and related elements in the absence of induction.在没有诱导的情况下，VanS传感器负向调控VanR介导的Tn1546及相关元件的糖肽抗性基因的转录激活。
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Diversity of VanA glycopeptide resistance elements in enterococci from humans and nonhuman sources.来自人类和非人类来源的肠球菌中VanA糖肽抗性元件的多样性。
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Diversity of Tn1546 in vanA-positive Enterococcus faecium clinical isolates with VanA, VanB, and VanD phenotypes and susceptibility to vancomycin.Tn1546 多样性在 VanA、VanB 和 VanD 表型以及对万古霉素敏感性的 VanA 阳性屎肠球菌临床分离株中。
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VanB phenotype-vanA genotype Enterococcus faecium with heterogeneous expression of teicoplanin resistance.具有替考拉宁耐药性异质性表达的VanB表型-vanA基因型屎肠球菌。
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Diversity of Tn1546 and its role in the dissemination of vancomycin-resistant enterococci in Portugal.Tn1546的多样性及其在葡萄牙耐万古霉素肠球菌传播中的作用。
Antimicrob Agents Chemother. 2008 Mar;52(3):1001-8. doi: 10.1128/AAC.00999-07. Epub 2008 Jan 7.
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Molecular analysis of Tn1546 in vanA-containing Enterococcus spp. isolated from humans and poultry.从人和家禽中分离出的含vanA的肠球菌属中Tn1546的分子分析。
Antimicrob Agents Chemother. 2000 Sep;44(9):2588-9. doi: 10.1128/AAC.44.9.2588-2589.2000.
4
PCR fragment length polymorphism analysis of vancomycin-resistant Enterococcus faecium.耐万古霉素屎肠球菌的聚合酶链反应片段长度多态性分析
J Clin Microbiol. 2000 Aug;38(8):2885-8. doi: 10.1128/JCM.38.8.2885-2888.2000.

本文引用的文献

1
Molecular analysis of diverse elements mediating VanA glycopeptide resistance in enterococci.介导肠球菌VanA糖肽抗性的多种因素的分子分析。
J Antimicrob Chemother. 1998 Nov;42(5):605-12. doi: 10.1093/jac/42.5.605.
2
Two-component signal-transducing systems involved in stress responses and vancomycin susceptibility in Lactobacillus sakei.参与清酒乳杆菌应激反应和万古霉素敏感性的双组分信号转导系统。
Microbiology (Reading). 1998 Oct;144 ( Pt 10):2873-2883. doi: 10.1099/00221287-144-10-2873.
3
Long PCRs of transposons in the structural analysis of genes encoding acquired glycopeptide resistance in enterococci.用于肠球菌中获得性糖肽抗性编码基因结构分析的转座子长链聚合酶链式反应
Biotechniques. 1998 Mar;24(3):432-7. doi: 10.2144/98243st02.
4
Diversity of VanA glycopeptide resistance elements in enterococci from humans and nonhuman sources.来自人类和非人类来源的肠球菌中VanA糖肽抗性元件的多样性。
Antimicrob Agents Chemother. 1998 Mar;42(3):502-8. doi: 10.1128/AAC.42.3.502.
5
Identification and characterization of IS1476, an insertion sequence-like element that disrupts VanY function in a vancomycin-resistant Enterococcus faecium strain.IS1476的鉴定与特性分析，IS1476是一种插入序列样元件，可破坏一株耐万古霉素屎肠球菌中VanY的功能。
Antimicrob Agents Chemother. 1997 Aug;41(8):1805-7. doi: 10.1128/AAC.41.8.1805.
6
The VanS sensor negatively controls VanR-mediated transcriptional activation of glycopeptide resistance genes of Tn1546 and related elements in the absence of induction.在没有诱导的情况下，VanS传感器负向调控VanR介导的Tn1546及相关元件的糖肽抗性基因的转录激活。
J Bacteriol. 1997 Jan;179(1):97-106. doi: 10.1128/jb.179.1.97-106.1997.
7
Characterization of Tn1546, a Tn3-related transposon conferring glycopeptide resistance by synthesis of depsipeptide peptidoglycan precursors in Enterococcus faecium BM4147.Tn1546的特性分析，Tn1546是一种与Tn3相关的转座子，通过在屎肠球菌BM4147中合成脂肽类肽聚糖前体赋予糖肽抗性。
J Bacteriol. 1993 Jan;175(1):117-27. doi: 10.1128/jb.175.1.117-127.1993.
8
Heterogeneity of the vanA gene cluster in clinical isolates of enterococci from the northeastern United States.美国东北部肠球菌临床分离株中vanA基因簇的异质性
Antimicrob Agents Chemother. 1995 Feb;39(2):362-8. doi: 10.1128/AAC.39.2.362.
9
The VanS-VanR two-component regulatory system controls synthesis of depsipeptide peptidoglycan precursors in Enterococcus faecium BM4147.VanS-VanR双组分调控系统控制着屎肠球菌BM4147中脂肽聚糖前体的合成。
J Bacteriol. 1992 Apr;174(8):2582-91. doi: 10.1128/jb.174.8.2582-2591.1992.

Disruption of vanS by IS1216V in a clinical isolate of Enterococcus faecium with VanA glycopeptide resistance.

作者信息

Darini A L, Palepou M F, James D, Woodford N

出版信息

Antimicrob Agents Chemother. 1999 Apr;43(4):995-6. doi: 10.1128/AAC.43.4.995.

DOI:10.1128/AAC.43.4.995
PMID:10200871
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC89249/
Abstract
摘要