EBNA-1 sequences in endemic and sporadic Burkitt's lymphoma.
作者信息
Bhatia K, Magrath I
出版信息
J Virol. 1999 Aug;73(8):7096-7.
Abstract
摘要
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EBNA-1 sequences in endemic and sporadic Burkitt's lymphoma.地方性和散发性伯基特淋巴瘤中的EBNA-1序列
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Carcinoma-risk variant of EBNA1 deregulates Epstein-Barr Virus episomal latency.EBNA1的癌风险变体可解除爱泼斯坦-巴尔病毒游离体潜伏期的调控。
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本文引用的文献
1
Epstein-barr virus nuclear antigen 1 sequences in endemic and sporadic Burkitt's lymphoma reflect virus strains prevalent in different geographic areas.地方性和散发性伯基特淋巴瘤中的爱泼斯坦-巴尔病毒核抗原1序列反映了不同地理区域流行的病毒株。
J Virol. 1999 Feb;73(2):965-75. doi: 10.1128/JVI.73.2.965-975.1999.
2
Diversity of naturally occurring Epstein-Barr virus revealed by nucleotide sequence polymorphism in hypervariable domains in the BamHI K and N subgenomic regions.通过BamHI K和N亚基因组区域高变域中的核苷酸序列多态性揭示的自然发生的爱泼斯坦-巴尔病毒的多样性
J Gen Virol. 1998 Nov;79 ( Pt 11):2809-17. doi: 10.1099/0022-1317-79-11-2809.
3
4
5
Evolutionary dynamics of genetic variation in Epstein-Barr virus isolates of diverse geographical origins: evidence for immune pressure-independent genetic drift.不同地理来源的爱泼斯坦-巴尔病毒分离株中基因变异的进化动力学：免疫压力非依赖性基因漂变的证据
J Virol. 1997 Nov;71(11):8340-6. doi: 10.1128/JVI.71.11.8340-8346.1997.
6
Sequence variations in EBNA-1 may dictate restriction of tissue distribution of Epstein-Barr virus in normal and tumour cells.EBNA-1中的序列变异可能决定了爱泼斯坦-巴尔病毒在正常细胞和肿瘤细胞中的组织分布限制。
J Gen Virol. 1997 Jul;78 ( Pt 7):1663-70. doi: 10.1099/0022-1317-78-7-1663.
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8
Alterations in the structure of the EBV nuclear antigen, EBNA1, in epithelial cell tumours.上皮细胞瘤中EB病毒核抗原EBNA1的结构改变
Oncogene. 1995 Apr 20;10(8):1545-52.
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T cell responses and virus evolution: loss of HLA A11-restricted CTL epitopes in Epstein-Barr virus isolates from highly A11-positive populations by selective mutation of anchor residues.T细胞反应与病毒进化：通过锚定残基的选择性突变，在来自A11高度阳性人群的爱泼斯坦-巴尔病毒分离株中HLA A11限制性CTL表位的缺失
J Exp Med. 1994 Apr 1;179(4):1297-305. doi: 10.1084/jem.179.4.1297.
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EBV strain variation: geographical distribution and relation to disease state.
Virology. 1992 Sep;190(1):168-75. doi: 10.1016/0042-6822(92)91202-6.
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