致命性心肌再灌注损伤：它是否存在，我们是否应该对其进行治疗？ - Suppr

Lethal Myocardial Reperfusion Injury: Does it Exist, Should We Treat It?

作者信息

Ambrosio G, Tritto I

机构信息

Division of Cardiology. University of Perugia School of Medicine, Perugia, Italy.

出版信息

J Thromb Thrombolysis. 1997 Jan;4(1):69-70. doi: 10.1023/a:1017554619252.

DOI:10.1023/a:1017554619252

PMID:10639229

Abstract

摘要

相似文献

Lethal Myocardial Reperfusion Injury: Does it Exist, Should We Treat It?

J Thromb Thrombolysis. 1997 Jan;4(1):69-70. doi: 10.1023/a:1017554619252.

Involvement of neutrophils in the pathogenesis of lethal myocardial reperfusion injury.

Cardiovasc Res. 2004 Feb 15;61(3):481-97. doi: 10.1016/j.cardiores.2003.10.011.

Does reperfusion injury exist in humans?

J Am Coll Cardiol. 1993 Feb;21(2):537-45. doi: 10.1016/0735-1097(93)90700-b.

Pharmacological approaches to reperfusion therapy.

Cardiovasc Res. 2012 May 1;94(2):246-52. doi: 10.1093/cvr/cvs114. Epub 2012 Apr 18.

The Antioxidant, N-(2-mercaptopropionyl)-glycine (MPG), Does Not Reduce Myocardial Infarct Size in an Acute Canine Model of Myocardial Ischemia and Reperfusion.

J Thromb Thrombolysis. 1998 May;5(2):135-141. doi: 10.1023/A:1008830129106.

Progression in attenuating myocardial reperfusion injury: an overview.

Int J Cardiol. 2014 Jan 1;170(3):261-9. doi: 10.1016/j.ijcard.2013.11.007. Epub 2013 Nov 12.

Lethal myocardial reperfusion injury: a necessary evil?

Int J Cardiol. 2011 Aug 18;151(1):3-11. doi: 10.1016/j.ijcard.2010.10.056. Epub 2010 Nov 19.

Pre-conditioning and postconditioning to limit ischemia-reperfusion-induced myocardial injury: what could be the next footstep?

Pharmacol Res. 2008 Jun;57(6):403-12. doi: 10.1016/j.phrs.2008.05.006. Epub 2008 Jun 30.

Carvedilol improves function and reduces infarct size in the feline myocardium by protecting against lethal reperfusion injury.

Eur J Pharmacol. 1996 Oct 24;314(1-2):99-107. doi: 10.1016/s0014-2999(96)00549-3.

Ischemia, reperfusion, and the determinants of tissue injury.

Cardiovasc Drugs Ther. 1990 Aug;4 Suppl 4:767-76. doi: 10.1007/BF00051274.

引用本文的文献

Human Heart Anoxia and Reperfusion Tissue (HEART) Model for the Rapid Study of Exosome Bound miRNA Expression As Biomarkers for Myocardial Infarction.

Small. 2022 Jul;18(28):e2201330. doi: 10.1002/smll.202201330. Epub 2022 Jun 7.

Downregulation of miR‑16 protects H9c2(2‑1) cells against hypoxia/reoxygenation damage by targeting CIAPIN1 and regulating the NF‑κB pathway.

Mol Med Rep. 2019 Oct;20(4):3113-3122. doi: 10.3892/mmr.2019.10568. Epub 2019 Aug 7.

Treatment with the C5a receptor antagonist ADC-1004 reduces myocardial infarction in a porcine ischemia-reperfusion model.

BMC Cardiovasc Disord. 2010 Sep 27;10:45. doi: 10.1186/1471-2261-10-45.

Apyrase treatment of myocardial infarction according to a clinically applicable protocol fails to reduce myocardial injury in a porcine model.

BMC Cardiovasc Disord. 2010 Jan 4;10:1. doi: 10.1186/1471-2261-10-1.

Mild hypothermia markedly reduces ischemia related coronary t-PA release.

J Thromb Thrombolysis. 2010 Apr;29(3):289-95. doi: 10.1007/s11239-009-0350-2.

Rapid short-duration hypothermia with cold saline and endovascular cooling before reperfusion reduces microvascular obstruction and myocardial infarct size.

BMC Cardiovasc Disord. 2008 Apr 10;8:7. doi: 10.1186/1471-2261-8-7.

本文引用的文献

Modulation of platelet function by reactive oxygen metabolites.

Am J Physiol. 1994 Jul;267(1 Pt 2):H308-18. doi: 10.1152/ajpheart.1994.267.1.H308.

Superoxide radical: controversies, contradictions, and paradoxes.

Proc Soc Exp Biol Med. 1995 Jun;209(2):112-7. doi: 10.3181/00379727-209-43885c.

The role of oxygen free radicals in preconditioning.

J Mol Cell Cardiol. 1995 Apr;27(4):1035-9. doi: 10.1016/0022-2828(95)90072-1.

Reduction in experimental infarct size by recombinant human superoxide dismutase: insights into the pathophysiology of reperfusion injury.

Circulation. 1986 Dec;74(6):1424-33. doi: 10.1161/01.cir.74.6.1424.

Free radicals and myocardial ischemia and reperfusion injury.

J Lab Clin Med. 1987 Jul;110(1):13-30.

"Reperfusion injury" by oxygen-derived free radicals? Effect of superoxide dismutase plus catalase, given at the time of reperfusion, on myocardial infarct size, contractile function, coronary microvasculature, and regional myocardial blood flow.

Circ Res. 1989 Jan;64(1):86-96. doi: 10.1161/01.res.64.1.86.

The role of neutrophils and free radicals in the ischemic-reperfused heart: why the confusion and controversy?

J Mol Cell Cardiol. 1989 Dec;21(12):1225-39. doi: 10.1016/0022-2828(89)90669-x.

Can superoxide dismutase alter myocardial infarct size?

Circulation. 1989 May;79(5):1137-42. doi: 10.1161/01.cir.79.5.1137.

Reperfusion injury and its pharmacologic modification.

Circulation. 1989 Oct;80(4):1049-62. doi: 10.1161/01.cir.80.4.1049.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验

Lethal Myocardial Reperfusion Injury: Does it Exist, Should We Treat It?

作者信息

机构信息

出版信息

相似文献

引用本文的文献

本文引用的文献

文献AI研究员

用中文搜PubMed

文档翻译

Suppr 超能文献

相似文献

引用本文的文献

本文引用的文献