Mira Y, García del Castillo G, Estellés A, Villa P, Vayá A, Ortuño C, Aznar J
Department of Clinical Pathology, La Fe Hospital University, Valencia, Spain.
Clin Appl Thromb Hemost. 2000 Jul;6(3):179-80. doi: 10.1177/107602960000600311.
We describe a patient with left branch portal vein thrombosis involving two thrombophilic alterations, the prothrombin G20210A mutation and protein C deficiency. In spite of not being under anticoagulant treatment, the thrombus in the portal vein underwent complete and spontaneous lysis. No other risk factors were detected and no family history related to thrombosis was found.
我们描述了一名患有左支门静脉血栓形成的患者，该患者存在两种易栓改变，即凝血酶原G20210A突变和蛋白C缺乏。尽管未接受抗凝治疗，但门静脉内的血栓仍完全自发溶解。未检测到其他危险因素，也未发现与血栓形成相关的家族史。
