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非酮症高血糖伴偏身投掷-偏身舞蹈症患者T1加权磁共振成像显示同侧壳核高信号：一例报告

Ipsilateral putamen hyperintensity on T1-weighted MRI in non-ketotic hyperglycemia with hemiballism-hemichorea: a case report.

作者信息

Lin J J.

机构信息

Department of Neurology, Chushang Show-Chwan Hospital, No. 75, Sec. 2, Chi-Shang Road, Chushang Jenn, 557, R.O.C., Nantou, Taiwan

出版信息

Parkinsonism Relat Disord. 2001 Oct;7(4):319-321. doi: 10.1016/s1353-8020(00)00072-9.

DOI:10.1016/s1353-8020(00)00072-9
PMID:11344017
Abstract

Movement disorders are often caused by lesions in the contralateral basal ganglia. This report presents an elderly woman with hemiballism-hemichorea induced by non-ketotic hyperglycemia, whose brain CT revealed a hyperdense lesion over the ipsilateral putamen. Concomitantly, MR image revealed a putaminal hyperintensity on T1-weighted image and hypointensity on T2-weighted image. A follow-up brain CT 4months after remission of the hyperkinesia revealed that the abnormal intensity had been completely resolved.

摘要

运动障碍通常由对侧基底神经节病变引起。本报告介绍了一位老年女性，她因非酮症高血糖诱发偏侧投掷症-偏侧舞蹈症，其脑部CT显示同侧壳核有高密度病变。同时，磁共振成像显示壳核在T1加权图像上呈高信号，在T2加权图像上呈低信号。运动障碍缓解4个月后的随访脑部CT显示，异常信号已完全消失。

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