内质网和线粒体对神经元中钙离子处理的不同贡献。 - Suppr

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超能文献

Contrasting contributions of endoplasmic reticulum and mitochondria to Ca(2)+ handling in neurons.

作者信息

Barrett E F

机构信息

Department of Physiology and Biophysics, Neuroscience Program, Miami, FL 33101, USA.

出版信息

J Gen Physiol. 2001 Jul;118(1):79-82. doi: 10.1085/jgp.118.1.79.

DOI:10.1085/jgp.118.1.79

PMID:11429445

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2233750/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f557/2233750/aa0c6367e064/JGP8449.f1.jpg

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Stimulation-evoked increases in cytosolic [Ca(2+)] in mouse motor nerve terminals are limited by mitochondrial uptake and are temperature-dependent.刺激诱发的小鼠运动神经末梢胞质中[Ca(2+)]的增加受到线粒体摄取的限制，且具有温度依赖性。

J Neurosci. 2000 Oct 1;20(19):7290-6. doi: 10.1523/JNEUROSCI.20-19-07290.2000.

Mitochondria shape hormonally induced cytoplasmic calcium oscillations and modulate exocytosis.线粒体塑造激素诱导的细胞质钙振荡并调节胞吐作用。

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J Biol Chem. 2000 May 19;275(20):15305-13. doi: 10.1074/jbc.275.20.15305.

Dissection of mitochondrial Ca2+ uptake and release fluxes in situ after depolarization-evoked [Ca2+](i) elevations in sympathetic neurons.在交感神经元去极化诱发细胞内钙离子浓度升高后，原位剖析线粒体钙离子摄取和释放通量。

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