Royal H D, Shields J B, Donati R M
Arch Intern Med. 1975 Nov;135(11):1502-5.
Two instances of misplacement of a central venous pressure monitoring catheter into a pulmonary artery are presented. In one case, reported in detail, unilateral pulmonary edema developed during administration of 0.5N saline for the treatment of diabetic acidosis via the misplaced catheter, and it reverted following repositioning of the catheter in the superior vena caca. Experiments were carried out in dogs to explore possible causes of this phenomenon. The results of these experiments suggest that the release of vasoactive substances, stimulation of neural reflexes, or both are the pathophysiological mechanisms responsible for the development of the pulmonary edema.
本文报告了两例中心静脉压监测导管误置入肺动脉的病例。其中一例详细报道如下：在通过误置入的导管输注0.5N盐水治疗糖尿病酸中毒期间，出现了单侧肺水肿，导管重新置于上腔静脉后肺水肿消退。对犬进行了实验以探究此现象的可能原因。这些实验结果表明，血管活性物质的释放、神经反射的刺激或两者共同作用是导致肺水肿发生的病理生理机制。
