Otsuki Mika, Soma Yoshiaki, Arihiro Shoji, Watanabe Yoshimasa, Moriwaki Hiroshi, Naritomi Hiroaki
Cerebrovascular Division, Department of Internal Medicine, National Cardiovascular Center, Suita, Japan.
Eur Neurol. 2002;47(3):136-40. doi: 10.1159/000047971.
We report a 60-year-old right-handed Japanese man who showed an isolated persistent typing impairment without aphasia, agraphia, apraxia or any other neuropsychological deficit. We coined the term 'dystypia' for this peculiar neuropsychological manifestation. The symptom was caused by an infarction in the left frontal lobe involving the foot of the second frontal convolution and the frontal operculum. The patient's typing impairment was not attributable to a disturbance of the linguistic process, since he had no aphasia or agraphia. The impairment was not attributable to the impairment of the motor execution process either, since he had no apraxia. Thus, his typing impairment was deduced to be based on a disturbance of the intermediate process where the linguistic phonological information is converted into the corresponding performance. We hypothesized that there is a specific process for typing which branches from the motor programming process presented in neurolinguistic models. The foot of the left second frontal convolution and the operculum may play an important role in the manifestation of 'dystypia'.
我们报告了一名60岁的右利手日本男性,他表现出孤立的持续性打字障碍,无失语、失写、失用或任何其他神经心理学缺陷。我们将这种特殊的神经心理学表现称为“打字障碍”。该症状由左侧额叶梗死引起,累及第二额回的足部和额盖。患者的打字障碍并非归因于语言加工过程的紊乱,因为他没有失语或失写。该障碍也不归因于运动执行过程的损害,因为他没有失用。因此,推断他的打字障碍是基于语言音韵信息转化为相应行为的中间过程的紊乱。我们假设存在一个从神经语言模型中提出的运动编程过程分支出来的特定打字过程。左侧第二额回的足部和额盖可能在“打字障碍”的表现中起重要作用。