Myocardial dysfunction in sepsis: no role for NO?
作者信息
Belcher E, Mitchell J, Evans T
出版信息
Heart. 2002 Jun;87(6):507-9. doi: 10.1136/heart.87.6.507.
Abstract
摘要
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Heart. 2002 Jun;87(6):507-9. doi: 10.1136/heart.87.6.507.
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本文引用的文献
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Multiple-center, randomized, placebo-controlled, double-blind study of the nitric oxide synthase inhibitor 546C88: effect on survival in patients with septic shock.一氧化氮合酶抑制剂546C88的多中心、随机、安慰剂对照、双盲研究：对感染性休克患者生存率的影响
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Biosynthesis of constitutive nitric oxide synthase-derived nitric oxide attenuates coronary vasoconstriction and myocardial depression in a model of septic heart failure induced by Staphylococcus aureus alpha-toxin.
Crit Care Med. 2001 Jan;29(1):1-7. doi: 10.1097/00003246-200101000-00001.
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Nitric oxide, nitrates and ischaemic preconditioning.一氧化氮、硝酸盐与缺血预处理
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4
Inhibition of nitric oxide synthase augments the positive inotropic effect of nitric oxide donors in the rat heart.一氧化氮合酶的抑制增强了一氧化氮供体对大鼠心脏的正性肌力作用。
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Pharmacologic enhancement of tolerance to ischemic cardiac stress using monophosphoryl lipid A. A comparison with antecedent ischemia.
Ann N Y Acad Sci. 1999 Jun 30;874:222-35. doi: 10.1111/j.1749-6632.1999.tb09238.x.
6
An open-label dose escalation study of the nitric oxide synthase inhibitor, N(G)-methyl-L-arginine hydrochloride (546C88), in patients with septic shock. Glaxo Wellcome International Septic Shock Study Group.一项关于一氧化氮合酶抑制剂盐酸N(G)-甲基-L-精氨酸（546C88）用于感染性休克患者的开放标签剂量递增研究。葛兰素威康国际感染性休克研究小组。
Crit Care Med. 1999 May;27(5):913-22. doi: 10.1097/00003246-199905000-00025.
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Has the mortality of septic shock changed with time.感染性休克的死亡率随时间有变化吗？
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Inhaled nitric oxide prevents left ventricular impairment during endotoxemia.吸入一氧化氮可预防内毒素血症期间的左心室损伤。
J Appl Physiol (1985). 1998 Dec;85(6):2018-24. doi: 10.1152/jappl.1998.85.6.2018.
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In vivo gene transfection of human endothelial cell nitric oxide synthase in cardiomyocytes causes apoptosis-like cell death. Identification using Sendai virus-coated liposomes.心肌细胞中人类内皮细胞一氧化氮合酶的体内基因转染导致凋亡样细胞死亡。使用仙台病毒包被脂质体进行鉴定。
Circulation. 1997 May 20;95(10):2441-7. doi: 10.1161/01.cir.95.10.2441.
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