Ohkubo Kimie, Watanabe Ichiro, Kojima Toshiaki, Masaki Riko, Oshikawa Naohiro, Sugimura Hidezou, Okumura Yasuo, Yamada Takeshi, Saito Satoshi, Ozawa Yukio, Kanmatsuse Katsuo
Second Department of Medicine, Nihon University School of Medicine, Tokyo, Japan.
Pacing Clin Electrophysiol. 2002 Jun;25(6):986-8. doi: 10.1046/j.1460-9592.2002.00986.x.
The patient was a 40-year-old woman with a history of surgery for atrial septal defect and catheter ablation for typical atrial flutter. An electrophysiological study was performed because she had palpitation and syncope. She had ectopic atrial rhythm originating from low lateral RA. Two focal atrial tachycardias ([1] superior vena cava-RA junction and [2] a lowposteroseptal RA) were successfully ablated. Following catheter ablation for the second atrial tachycardia, she developed junctional rhythm because ectopic atrial rhythm showed exit block. However, atrial activation of junctional rhythm could conduct into the ectopic atrial rhythm focus and reset the rhythm when atrial activation of junctional rhythm reached the blocked line after atrial refractoriness by preceding ectopic atrial rhythm.
该患者为一名40岁女性,有房间隔缺损手术史及典型心房扑动导管消融史。因心悸和晕厥进行了电生理检查。她有起源于右下外侧右心房的异位心房节律。成功消融了两种局灶性房性心动过速([1]上腔静脉-右心房交界处和[2]右下后间隔右心房)。在对第二次房性心动过速进行导管消融后,她出现了交界性心律,因为异位心房节律出现了传出阻滞。然而,交界性心律的心房激动可传入异位心房节律灶,并在先前异位心房节律导致心房不应期后,当交界性心律的心房激动到达阻滞线时重置节律。