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相似文献

1
Enhancement of rIn:rCn-induced interferon production by amphotericin B.两性霉素B增强rIn:rCn诱导的干扰素产生。
Antimicrob Agents Chemother. 1976 Mar;9(3):551-3. doi: 10.1128/AAC.9.3.551.
2
Synergistic cytotoxicity for L929 cells of polyriboinosinic-polyribocytidylic acid and amphotericin B methyl ester.聚肌苷酸-聚胞苷酸与两性霉素B甲酯对L929细胞的协同细胞毒性。
J Natl Cancer Inst. 1978 Jun;60(6):1309-11. doi: 10.1093/jnci/60.6.1309.
3
Mechanistic studies of polyene enhancement of interferon production by polyriboinosinic-polyribocytidylic acid.多烯增强聚肌苷酸-聚胞苷酸诱导干扰素产生的机制研究。
Antimicrob Agents Chemother. 1978 Feb;13(2):159-64. doi: 10.1128/AAC.13.2.159.
4
Interferon induction in primates by stabilized polyriboinosinic acid-polyribocytidylic acid: effect of component size.稳定化聚肌苷酸-聚胞苷酸在灵长类动物中诱导干扰素：组分大小的影响
Infect Immun. 1981 Nov;34(2):416-21. doi: 10.1128/iai.34.2.416-421.1981.
5
Effects of amphotericin B and its methyl ester on the antiviral activity of polyinosinic:polycytidylic acid.两性霉素B及其甲酯对聚肌苷酸:聚胞苷酸抗病毒活性的影响。
Antimicrob Agents Chemother. 1979 Aug;16(2):203-9. doi: 10.1128/AAC.16.2.203.
6
Mechanisms of DEAE-dextran enhancement of polynucleotide induction of interferon.二乙氨基乙基葡聚糖增强多核苷酸诱导干扰素的机制。
Proc Soc Exp Biol Med. 1971 Apr;136(4):1111-4. doi: 10.3181/00379727-136-35440.
7
Increase by calcium in production of interferon by L929 cells induced with polyriboinosinate-polyribocytidylate complex.聚肌苷酸-聚胞苷酸复合物诱导L929细胞产生干扰素过程中钙的增加。
J Gen Virol. 1978 Aug;40(2):485-8. doi: 10.1099/0022-1317-40-2-485.
8
Pharmacokinetics of interferon in blood, cerebrospinal fluid, and brain after administration of modified polyriboinosinic-polyribocytidylic acid and amphotericin B.给予修饰的聚肌苷酸-聚胞苷酸和两性霉素B后干扰素在血液、脑脊液及脑内的药代动力学
J Infect Dis. 1982 Dec;146(6):819-25. doi: 10.1093/infdis/146.6.819.
9
Molecular species of interferon induced in mouse L cells by Newcastle disease virus and polyriboinosinic-polyribocytidylic acid.新城疫病毒和聚肌苷酸-聚胞苷酸在小鼠L细胞中诱导产生的干扰素的分子种类。
J Gen Virol. 1979 Jun;43(3):521-9. doi: 10.1099/0022-1317-43-3-521.
10
Binding of polyriboinosinic-polyribocytidylic acid with cultured cells.聚肌苷酸-聚胞苷酸与培养细胞的结合
Arch Virol. 1975;49(2-3):229-37. doi: 10.1007/BF01317541.

引用本文的文献

1
Liposomal amphotericin B-the future.脂质体两性霉素 B——未来。
J Antimicrob Chemother. 2022 Nov 25;77(Suppl_2):ii21-ii34. doi: 10.1093/jac/dkac353.
2
It only takes one to do many jobs: Amphotericin B as antifungal and immunomodulatory drug.只需一种药物就能发挥多种作用：两性霉素B作为抗真菌和免疫调节药物。
Front Microbiol. 2012 Aug 8;3:286. doi: 10.3389/fmicb.2012.00286. eCollection 2012.
3
Effects of amphotericin B and its methyl ester on the antiviral activity of polyinosinic:polycytidylic acid.两性霉素B及其甲酯对聚肌苷酸:聚胞苷酸抗病毒活性的影响。
Antimicrob Agents Chemother. 1979 Aug;16(2):203-9. doi: 10.1128/AAC.16.2.203.

本文引用的文献

1
The specificity of interferon induction in chick embryo cells by helical RNA.螺旋状RNA在鸡胚细胞中诱导干扰素的特异性。
Proc Natl Acad Sci U S A. 1969 May;63(1):160-7. doi: 10.1073/pnas.63.1.160.
2
Preparation of sepharose-bound poly (rI:rC).琼脂糖结合的聚（rI：rC）的制备。
Biochem Biophys Res Commun. 1971 Oct 1;45(1):184-9. doi: 10.1016/0006-291x(71)90067-2.
3
Potentiation of drug effect by Tween 80 in Chinese hamster cells resistant to actinomycin D and daunomycin.吐温80对耐放线菌素D和柔红霉素的中国仓鼠细胞药物效应的增强作用。
Cancer Res. 1972 Jun;32(6):1195-200.
4
Antibiotic interaction with model membranes.抗生素与模型膜的相互作用。
Annu Rev Pharmacol. 1970;10:119-42. doi: 10.1146/annurev.pa.10.040170.001003.
5
Lack of uptake of intact polyriboinosinic-polyribocytidylic acid by cells.细胞对完整的聚肌苷酸-聚胞苷酸摄取不足。
Proc Soc Exp Biol Med. 1973 Nov;144(2):534-7. doi: 10.3181/00379727-144-37630.
6
Permeability control in animal cells by polyenes: a possibility.多烯对动物细胞通透性的控制：一种可能性。
Antimicrob Agents Chemother. 1973 Mar;3(3):441-3. doi: 10.1128/AAC.3.3.441.
7
Fungal sterols and the mode of action of the polyene antibiotics.真菌甾醇与多烯抗生素的作用方式
Adv Appl Microbiol. 1974;17(0):109-34. doi: 10.1016/s0065-2164(08)70556-2.
8
Uptake of Escherichia coli DNA into HeLa cells enhanced by amphotericin B.
Nature. 1974 Jul 26;250(464):323-5. doi: 10.1038/250323a0.
9
Potentiation of the antifungal effects of antibiotics by amphotericin B.两性霉素B增强抗生素的抗真菌作用。
Antimicrob Agents Chemother. 1972 Aug;2(2):61-5. doi: 10.1128/AAC.2.2.61.
10
On the anatomy of amphotericin B-cholesterol pores in lipid bilayer membranes.脂质双分子层膜中两性霉素B-胆固醇孔的解剖学研究
Kidney Int. 1973 Nov;4(5):337-45. doi: 10.1038/ki.1973.126.

两性霉素B增强rIn:rCn诱导的干扰素产生。

Enhancement of rIn:rCn-induced interferon production by amphotericin B.

作者信息

Borden E C, Leonhardt P H

出版信息

Antimicrob Agents Chemother. 1976 Mar;9(3):551-3. doi: 10.1128/AAC.9.3.551.

DOI:10.1128/AAC.9.3.551
PMID:1259412
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC429570/
Abstract

When mouse L929 cells are treated with amphotericin B before exposure to polyriboinosinic-polyribocytidylic acid or polyriboinosinic-polyribocytidylic acid-dextran, they make significantly more interferon than do cells not receiving amphotericin. This effect may be due to enhanced cell membrane penetration by the polynucleotide.

摘要

当小鼠L929细胞在暴露于聚肌苷酸-聚胞苷酸或聚肌苷酸-聚胞苷酸-葡聚糖之前用两性霉素B处理时，它们产生的干扰素比未接受两性霉素的细胞显著更多。这种效应可能是由于多核苷酸增强了细胞膜的穿透性。