Kulbertus H E, de Laval-Rutten F, Casters P
Br Heart J. 1976 Jun;38(6):549-57. doi: 10.1136/hrt.38.6.549.
Aberrant ventricular conduction was induced in 44 subjects by introduction of atrial premature beats through a transvenous catheter-electrode. Multiple patterns of aberrant ventricular conduction were obtained in 32 patients and, in the whole group, 116 different configurations were recorded. Of these, 104 showed a classical pattern of mono- or biventricular conduction disturbance. The pattern frequencies were as follows: right bundle-branch block, 28; left anterior hemiblock combined with right bundle-branch block, 21; left anterior hemiblock, 17; left posterior hemiblock combined with right bundle-branch block, 12; left posterior hemiblock, 10; complete left bundle-branch block, 10; and incomplete left bundle-branch block, 6. The remaining 12 configurations could not be classified into the usual categories of intraventricular blocks. In 7 of them, the alterations only consisted of trivial modifications of the QRS contour. In the other 5 instances, aberrant conduction manifested itself by a conspicuous anterior displacement of the QRS loop, with increased duration of anterior forces. The latter observation is worthy of notice, as it indicates that, in the differential diagnosis of the vectorcardiographic pattern characterized by prominent anterior forces, conduction disturbances should be considered a possible aetiological factor in addition to right ventricular hypertrophy, and true posterior wall myocardial infarction.
通过经静脉导管电极引入房性早搏,在44名受试者中诱发了异常心室传导。32例患者获得了多种异常心室传导模式,在整个研究组中,记录到116种不同的形态。其中,104种表现为典型的单心室或双心室传导障碍模式。模式频率如下:右束支传导阻滞,28例;左前分支阻滞合并右束支传导阻滞,21例;左前分支阻滞,17例;左后分支阻滞合并右束支传导阻滞,12例;左后分支阻滞,10例;完全性左束支传导阻滞,10例;不完全性左束支传导阻滞,6例。其余12种形态无法归类为常见的室内传导阻滞类型。其中7例,改变仅表现为QRS波群形态的轻微改变。在另外5例中,异常传导表现为QRS环明显向前移位,前向力持续时间延长。后一观察结果值得注意,因为它表明,在以明显前向力为特征的心电图模式的鉴别诊断中,除了右心室肥厚和真正的后壁心肌梗死外,传导障碍应被视为可能的病因。