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[Studies on encephalitis due to human trypanosomiasis in Africa; clinical, anatomical, electroencephalographic, and biological correlations].[非洲人类锥虫病所致脑炎的研究；临床、解剖、脑电图及生物学相关性]
Med Trop (Mars). 1952 Nov-Dec;12(6):633-75.
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Trypanosoma brucei brucei crosses the blood-brain barrier while tight junction proteins are preserved in a rat chronic disease model.在大鼠慢性疾病模型中，布氏布氏锥虫可穿越血脑屏障，而紧密连接蛋白得以保留。
Neuropathol Appl Neurobiol. 2001 Feb;27(1):77-85. doi: 10.1046/j.0305-1846.2001.00306.x.
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Human African trypanosomiasis: MRI.人类非洲锥虫病：磁共振成像
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Chronic Gambian trypanosomiasis.慢性冈比亚锥虫病
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[Pseudo-tumoral human African trypanosomiasis due to Trypanosoma gambiense. Clinical and tomodensitometry study (author's transl)].冈比亚锥虫所致假性肿瘤型人类非洲锥虫病。临床与体层X线密度测定研究（作者译）
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Spread of Trypanosoma brucei to the nervous system: early attack on circumventricular organs and sensory ganglia.布氏锥虫向神经系统的传播：对室周器官和感觉神经节的早期侵袭。
J Neurosci Res. 1988 Sep;21(1):56-61. doi: 10.1002/jnr.490210109.
9
Epidemiology and control of African trypanosomiasis. Report of a WHO Expert Committee.非洲锥虫病的流行病学与控制。世界卫生组织专家委员会报告。
World Health Organ Tech Rep Ser. 1986;739:1-127.

非洲锥虫病的磁共振成像表现。

MR imaging findings in African trypansomiasis.

作者信息

Gill Danny S, Chatha Deep S, del Carpio-O'Donovan Raquel

机构信息

Department of Radiology, Montreal General Hospital, McGill University Health Center, Montreal, Quebec, Canada.

出版信息

AJNR Am J Neuroradiol. 2003 Aug;24(7):1383-5.

PMID:12917133
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7973687/
Abstract

We report the initial and follow-up brain findings in a 42-year-old male patient with CNS involvement with African trypansomiasis. Initial MR imaging demonstrated diffuse hyperintensity in the basal ganglia bilaterally as well as involvement of the internal capsule, external capsule, and extreme capsule. Follow-up examination at 1 year revealed decreased signal intensity in the previously affected areas; however, ventricular enlargement indicative of atrophy was readily apparent.

摘要

我们报告了一名42岁男性中枢神经系统受累的非洲锥虫病患者的初始及随访脑部检查结果。初始磁共振成像显示双侧基底节区弥漫性高信号，以及内囊、外囊和最外囊受累。1年后的随访检查显示先前受累区域信号强度降低；然而，脑室扩大提示萎缩很明显。