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[Congophile angiopathy and senile plaques in aged dogs].

作者信息

VON BRAUNMUHL A

出版信息

Arch Psychiatr Nervenkr Z Gesamte Neurol Psychiatr. 1956;194(4):396-414. doi: 10.1007/BF00350248.

DOI:10.1007/BF00350248
PMID:13340979
Abstract
摘要

相似文献

1
[Congophile angiopathy and senile plaques in aged dogs].
Arch Psychiatr Nervenkr Z Gesamte Neurol Psychiatr. 1956;194(4):396-414. doi: 10.1007/BF00350248.
2
Pathological studies on cerebral amyloid angiopathy, senile plaques and amyloid deposition in visceral organs in aged dogs.
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3
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4
[Histopathological studies on senile plaques and cerebral amyloid angiopathy in aged cynomolgus monkeys].
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5
Dementia in hereditary cerebral hemorrhage with amyloidosis-Dutch type is associated with cerebral amyloid angiopathy but is independent of plaques and neurofibrillary tangles.荷兰型遗传性脑出血伴淀粉样变性中的痴呆与脑淀粉样血管病相关，但与斑块和神经原纤维缠结无关。
Ann Neurol. 2001 Dec;50(6):765-72. doi: 10.1002/ana.10040.
6
Double-labeling immunohistochemical studies on canine senile plaques and cerebral amyloid angiopathy.
J Vet Med Sci. 1993 Aug;55(4):637-42. doi: 10.1292/jvms.55.637.
7
A retrospective study of canine senile plaques and cerebral amyloid angiopathy.
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[Electron microscopical studies of cerebral amyloidosis in aged dogs and a senile human].[老年犬和老年人类大脑淀粉样变性的电子显微镜研究]
Acta Neuropathol. 1971;19(2):129-36. doi: 10.1007/BF00688491.
9
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Acta Neuropathol. 1971;19(2):121-8. doi: 10.1007/BF00688490.
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Immunohistochemical studies on canine cerebral amyloid angiopathy and senile plaques.
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1
Vascular cognitive impairment: Modeling a critical neurologic disease in vitro and in vivo.血管性认知障碍：在体外和体内模拟一种关键的神经疾病。
Biochim Biophys Acta. 2016 May;1862(5):975-82. doi: 10.1016/j.bbadis.2015.12.009. Epub 2015 Dec 17.
2
Neurobiology of the aging dog.衰老犬的神经生物学
Age (Dordr). 2011 Sep;33(3):485-96. doi: 10.1007/s11357-010-9183-3. Epub 2010 Sep 16.
3
Oxidative stress, aging, and central nervous system disease in the canine model of human brain aging.人类大脑衰老犬模型中的氧化应激、衰老与中枢神经系统疾病

本文引用的文献

1
[Senile vascular necrosis].[老年血管性坏死]
Arch Psychiatr Nervenkr Z Gesamte Neurol Psychiatr. 1950;185(5):571-83. doi: 10.1007/BF00341626.
2
[Pathology of the brain in presenility & senility].[早老性与衰老性大脑的病理学]
Yokohama Med Bull. 1954 Aug;5(4):287-303.
3
[A particular type of senile angiopathy of the central nervous system: congophilic angiopathy, topography and frequency].[一种特殊类型的中枢神经系统老年血管病：嗜刚果红血管病，部位及发生率]
Vet Clin North Am Small Anim Pract. 2008 Jan;38(1):167-78, vi. doi: 10.1016/j.cvsm.2007.10.002.
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Neuritic plaque-like structures in the rat cerebellum following prolonged alcohol consumption.
Experientia. 1984 Jan 15;40(1):110-2. doi: 10.1007/BF01959131.
5
[Fluorescence microscopical observations on cerebral amyloidosis in aged dogs and senile humans].
Acta Neuropathol. 1971;19(2):121-8. doi: 10.1007/BF00688490.
6
Presenile dementia: some pathological problems and possibilites.早老性痴呆：一些病理学问题及可能性
Proc R Soc Med. 1971 Aug;64(8):829-31. doi: 10.1177/003591577106400811.
7
Thioflavin S fluorescent and Congo red anisotropic stainings in the histologic demonstration of amyloid.硫黄素S荧光染色和刚果红各向异性染色在淀粉样蛋白组织学显示中的应用
Acta Neuropathol. 1967 Feb 3;7(4):336-48. doi: 10.1007/BF00688089.
8
Ultrastructure of the microglia that phagocytose amyloid and the microglia that produce beta-amyloid fibrils.
Acta Neuropathol. 1992;84(3):225-33. doi: 10.1007/BF00227813.
9
Ultrastructural studies of the cells forming amyloid in the cortical vessel wall in Alzheimer's disease.阿尔茨海默病皮质血管壁中形成淀粉样蛋白的细胞的超微结构研究。
Acta Neuropathol. 1992;84(2):117-27. doi: 10.1007/BF00311383.
Monatsschr Psychiatr Neurol. 1954 Oct;128(4):219-56.
4
[Dementia pugilistica with anatomical findings].伴有解剖学发现的拳击性痴呆
Virchows Arch Pathol Anat Physiol Klin Med. 1954;325(6):680-709. doi: 10.1007/BF00955101.
5
[Polarization studies on the relation between nodular and fibrillary changes in the brain in Alzheimer's disease and nodular-like deposits of amyloid substance in various organs].
Dtsch Z Nervenheilkd. 1954;171(3):173-80.
6
[Focal paralysis with Alzheimer's change of fibrilles and primitive plaques].
Arch Psychiatr Nervenkr Z Gesamte Neurol Psychiatr. 1952;188(3):209-17. doi: 10.1007/BF00947040.