臭氧暴露后的生理、生化、免疫和病理变化。 - Suppr

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超能文献

Physiologic, biochemical, immunologic and pathologic changes following ozone exposure.

作者信息

SCHEEL L D, DOBROGORSKI O J, MOUNTAIN J T, SVIRBELY J L, STOKINGER H E

出版信息

J Appl Physiol. 1959 Jan;14(1):67-80. doi: 10.1152/jappl.1959.14.1.67.

DOI:10.1152/jappl.1959.14.1.67

PMID:13630829

Abstract

摘要

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Physiologic, biochemical, immunologic and pathologic changes following ozone exposure.臭氧暴露后的生理、生化、免疫和病理变化。

J Appl Physiol. 1959 Jan;14(1):67-80. doi: 10.1152/jappl.1959.14.1.67.

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Inconsequential role for chemerin-like receptor 1 in the manifestation of ozone-induced lung pathophysiology in male mice.Chemerin 样受体 1 在雄性小鼠臭氧诱导的肺病理生理学中的作用微不足道。

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Chemokine (C-C Motif) Receptor-Like 2 is not essential for lung injury, lung inflammation, or airway hyperresponsiveness induced by acute exposure to ozone.趋化因子（C-C基序）受体样2对于急性暴露于臭氧引起的肺损伤、肺部炎症或气道高反应性并非必不可少。

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Plasminogen activator inhibitor-1 does not contribute to the pulmonary pathology induced by acute exposure to ozone.纤溶酶原激活物抑制剂-1对急性暴露于臭氧引起的肺部病变没有作用。

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Human health effects of air pollution.空气污染对人类健康的影响。

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The effects of ozone on immune function.臭氧对免疫功能的影响。

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Erythrocyte survival in sheep exposed to ozone.暴露于臭氧环境下绵羊的红细胞存活情况。

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Exposure to ozone reduces influenza disease severity and alters distribution of influenza viral antigens in murine lungs.暴露于臭氧可降低流感疾病的严重程度，并改变流感病毒抗原在小鼠肺部的分布。

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In vivo inactivation of lysozyme by ozone.臭氧对溶菌酶的体内失活作用。

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Early response of lung to low levels of ozone.肺对低水平臭氧的早期反应。

Am J Pathol. 1974 Jan;74(1):31-58.

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