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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Takahashi K, Akiba Y, Horiguchi M

Department of Animal Science, Faculty of Agriculture, Tohoku University, Sendai Japan.

Comp Biochem Physiol C Comp Pharmacol Toxicol. 1992 May;102(1):73-5. doi: 10.1016/0742-8413(92)90046-a.

The experiments were undertaken to determine if an antithyroid agent (propylthiouracil, PTU) and/or ascorbic acid (AA) affect mixed-function oxidase (MFO) in hepatic microsomes of male broiler chicks. 2. Feeding PTU increased the MFO activity in a dose-related manner. Addition of AA to the PTU-containing diet further increased the content of cytochromes P-450 and b5, but not the activities of NADPH-cytochrome c and NADH-cytochrome b5 reductase, and the drug-metabolizing enzymes. 3. Supplemental AA induced cytochrome b5 rather than cytochrome P-450.

Takahashi K, Akiba Y, Horiguchi M

Department of Animal Science, Faculty of Agriculture, Tohoku University, Sendai Japan.

Comp Biochem Physiol C Comp Pharmacol Toxicol. 1992 May;102(1):73-5. doi: 10.1016/0742-8413(92)90046-a.

The experiments were undertaken to determine if an antithyroid agent (propylthiouracil, PTU) and/or ascorbic acid (AA) affect mixed-function oxidase (MFO) in hepatic microsomes of male broiler chicks. 2. Feeding PTU increased the MFO activity in a dose-related manner. Addition of AA to the PTU-containing diet further increased the content of cytochromes P-450 and b5, but not the activities of NADPH-cytochrome c and NADH-cytochrome b5 reductase, and the drug-metabolizing enzymes. 3. Supplemental AA induced cytochrome b5 rather than cytochrome P-450.