Ahmaidi S, Varray A, Collomp K, Mercier J, Préfaut C
Service d'Exploration Fonctionnelle Respiratoire, Hôpital Aiguelongue, Montpellier, France.
C R Seances Soc Biol Fil. 1992;186(1-2):145-55.
The time-course of heart rate, blood lactate, and ventilatory gas exchange was studied during an incremental exercise test on cycloergometer in order to ascertain whether heart rate deflection occurred at the same load as the second lactate S[La]2) and ventilatory (SV2) thresholds. Twelve moderately trained subjects, 22 to 30 years old, participated in the study. The initial power setting was 30 W for 3 min with successive increases of 30 W every min except at the end of the test where the increase was reduced to 20 and 10 W.min-1. Ventilatory flow (VE), oxygen uptake (VO2), carbon dioxide production (VCO2, ventilatory equivalents of O2 (EO2 = VE/VO2) and CO2 (ECO2 = VE/VCO2), and heart rate (HR) were determined during the last 20 s of every min. Venous blood samples were drawn at the end of each stage of effort and analyzed enzymatically for lactate concentration ([La]). The HR deflection, S[La]2, and SV2 were represented graphically by two investigators using a double blind procedure. Following the method proposed by Conconi et al. 1982, the deflection in HR was considered to begin at the point beyond which the increase in work intensity exceeded the increase in HR and the linearity of the work rate/HR relationship was lost. S[La]2 corresponded to the second breaking point of the lactate time-course curve (onset of blood lactate accumulation) and SV2 was identified at the second breaking point in the increase in VE and ventilatory equivalent for O2 uptake accompanied by a concomitant increase in ventilatory equivalent for CO2 output. We observed that the deflection point in HR was present only in 7 subjects. The work load, VO2, HR, and [La] levels at which heart rate departed from linearity did not differ significantly from those determined with S[La]2 ans SV2. The VO2 and HR values at HR deflection point were significantly correlated with those measured at S[La]2 and SV2. It is concluded that deflection in heart rate does not always occur, and when it does, it coincides with the second lactate and ventilatory gas exchange thresholds. It can thus be used for the determination of optimal intensity for individualized aerobic training.
为了确定心率偏移是否与第二次乳酸(S[La]2)和通气(SV2)阈值出现在相同负荷下,在自行车测力计上进行递增运动试验期间,研究了心率、血乳酸和通气气体交换的时程。12名年龄在22至30岁之间的中度训练受试者参与了该研究。初始功率设置为30 W,持续3分钟,之后每分钟连续增加30 W,但在试验结束时,增加幅度降至20和10 W·min-1。在每分钟的最后20秒测定通气流量(VE)、摄氧量(VO2)、二氧化碳产生量(VCO2)、O2的通气当量(EO2 = VE/VO2)和CO2的通气当量(ECO2 = VE/VCO2)以及心率(HR)。在每个运动阶段结束时采集静脉血样,并通过酶法分析乳酸浓度([La])。两名研究人员采用双盲程序以图形方式表示HR偏移、S[La]2和SV2。按照Conconi等人1982年提出的方法,心率偏移被认为从工作强度增加超过心率增加且工作率/心率关系的线性丧失的点开始。S[La]2对应于乳酸时程曲线的第二个转折点(血乳酸积累开始),SV2在VE和O2摄取通气当量增加的第二个转折点处确定,同时伴有CO2排出通气当量的相应增加。我们观察到,只有7名受试者出现了心率偏移点。心率偏离线性时的工作负荷、VO2、HR和[La]水平与用S[La]2和SV2确定的值没有显著差异。心率偏移点处的VO2和HR值与在S[La]2和SV2处测量的值显著相关。得出的结论是,心率偏移并非总是出现,当出现时,它与第二次乳酸和通气气体交换阈值一致。因此,它可用于确定个性化有氧训练的最佳强度。