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三尖瓣位人工瓣膜的多普勒超声心动图评估

Doppler echocardiographic evaluation of prosthetic valves in tricuspid position.

作者信息

Aoyagi Shigeaki, Tomoeda Hiroshi, Kawano Hiroshi, Yokose Shogo, Fukunaga Shuji

机构信息

Department of Surgery, Kurume University School of Medicine, Kurume, Japan.

出版信息

Asian Cardiovasc Thorac Ann. 2003 Sep;11(3):193-7. doi: 10.1177/021849230301100302.

Abstract

Doppler echocardiographic characteristics of 29 normally functioning prosthetic valves (23 mechanical, 6 biological) and 8 obstructed mechanical prostheses in the tricuspid position are reported. In normally functioning prostheses, peak velocity, mean pressure gradient, and pressure-half time were 1.25 +/- 0.18 m x sec(-1), 2.6 +/- 1.1 mm Hg, and 122.6 +/- 30.7 msec, respectively. Although no significant differences were seen in peak velocity and mean pressure gradient between mechanical and biological valves, the pressure half-time was significantly greater in biological valves. All normally functioning prostheses had a mean pressure gradient 5.5 mm Hg and pressure half-time < 200 msec. In obstructed bileaflet valves, peak velocity was 1.66 +/- 0.28 m x sec(-1), mean pressure gradient was 6.1 +/- 2.8 mm Hg, and pressure half-time was 265.8 +/- 171.7 msec. These Doppler data were significantly greater than those in normally functioning valves where the mean pressure gradient was 5.1 mm Hg and the pressure half-time was 156 msec in all except one patient. Pathological obstruction of a tricuspid prosthesis can be strongly suspected in patients with a mean pressure gradient > 5.5 mm Hg and a pressure half-time > 200 msec on Doppler echocardiography.

摘要

本文报告了29个功能正常的人工瓣膜(23个机械瓣,6个生物瓣)及8个三尖瓣位梗阻性机械瓣的多普勒超声心动图特征。功能正常的人工瓣膜,峰值流速、平均压力阶差和压力减半时间分别为1.25±0.18m·sec⁻¹、2.6±1.1mmHg和122.6±30.7msec。虽然机械瓣和生物瓣在峰值流速和平均压力阶差上无显著差异,但生物瓣的压力减半时间显著更长。所有功能正常的人工瓣膜平均压力阶差<5.5mmHg,压力减半时间<200msec。在梗阻性双叶瓣中,峰值流速为1.66±0.28m·sec⁻¹,平均压力阶差为6.1±2.8mmHg,压力减半时间为265.8±171.7msec。这些多普勒数据显著高于功能正常瓣膜,功能正常瓣膜平均压力阶差为5.1mmHg,除1例患者外压力减半时间为156msec。对于多普勒超声心动图检查平均压力阶差>5.5mmHg且压力减半时间>200msec的患者,强烈怀疑存在三尖瓣人工瓣膜的病理性梗阻。

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