Angiogenesis as a target in rheumatoid arthritis.
作者信息
Koch A E
机构信息
Feinberg School of Medicine, Northwestern University and Veteran's Administration Chicago Healthcare System, Lakeside Division, IL 60611, USA.
出版信息
Ann Rheum Dis. 2003 Nov;62 Suppl 2(Suppl 2):ii60-7. doi: 10.1136/ard.62.suppl_2.ii60.
Abstract
摘要
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本文引用的文献
1
Angiogenesis in arthritis: role in disease pathogenesis and as a potential therapeutic target.关节炎中的血管生成：在疾病发病机制中的作用及作为潜在治疗靶点
Angiogenesis. 1998;2(4):295-307. doi: 10.1023/a:1009229508096.
2
HIF-1alpha is essential for myeloid cell-mediated inflammation.低氧诱导因子-1α（HIF-1α）对于髓样细胞介导的炎症至关重要。
Cell. 2003 Mar 7;112(5):645-57. doi: 10.1016/s0092-8674(03)00154-5.
3
Angiopoietin-1 is expressed in the synovium of patients with rheumatoid arthritis and is induced by tumour necrosis factor alpha.血管生成素-1在类风湿性关节炎患者的滑膜中表达，并由肿瘤坏死因子α诱导产生。
Ann Rheum Dis. 2003 Feb;62(2):100-7. doi: 10.1136/ard.62.2.100.
4
Src and phosphatidylinositol 3-kinase mediate soluble E-selectin-induced angiogenesis.Src和磷脂酰肌醇3激酶介导可溶性E选择素诱导的血管生成。
Blood. 2003 May 15;101(10):3960-8. doi: 10.1182/blood-2002-04-1237. Epub 2003 Jan 9.
5
Inhibition of interleukin-1 but not tumor necrosis factor suppresses neovascularization in rat models of corneal angiogenesis and adjuvant arthritis.在角膜血管生成和佐剂性关节炎大鼠模型中，抑制白细胞介素-1而非肿瘤坏死因子可抑制新生血管形成。
Arthritis Rheum. 2002 Oct;46(10):2604-12. doi: 10.1002/art.10546.
6
Hypoxia-induced production of stromal cell-derived factor 1 (CXCL12) and vascular endothelial growth factor by synovial fibroblasts.缺氧诱导滑膜成纤维细胞产生基质细胞衍生因子1（CXCL12）和血管内皮生长因子。
Arthritis Rheum. 2002 Oct;46(10):2587-97. doi: 10.1002/art.10520.
7
Study of the mechanism involved in angiogenesis and synovial cell proliferation in human synovial tissues of patients with rheumatoid arthritis using SCID mice.利用重症联合免疫缺陷（SCID）小鼠对类风湿性关节炎患者人类滑膜组织中血管生成及滑膜细胞增殖所涉及机制的研究。
Lab Invest. 2002 Aug;82(8):981-8. doi: 10.1097/01.lab.0000022220.44511.40.
8
Cyclosporine inhibition of vascular endothelial growth factor production in rheumatoid synovial fibroblasts.环孢素对类风湿性滑膜成纤维细胞中血管内皮生长因子产生的抑制作用。
Arthritis Rheum. 2002 May;46(5):1202-9. doi: 10.1002/art.10215.
9
Revascularization of ischemic tissues by PlGF treatment, and inhibition of tumor angiogenesis, arthritis and atherosclerosis by anti-Flt1.通过胎盘生长因子（PlGF）治疗实现缺血组织的血管再生，以及通过抗Flt1抑制肿瘤血管生成、关节炎和动脉粥样硬化。
Nat Med. 2002 Aug;8(8):831-40. doi: 10.1038/nm731. Epub 2002 Jul 1.
10
Differential expression of the angiogenic Tie receptor family in arthritic and normal synovial tissue.血管生成性Tie受体家族在关节炎性和正常滑膜组织中的差异表达。
Arthritis Res. 2002;4(3):201-8. doi: 10.1186/ar407. Epub 2002 Jan 16.
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