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1
A mononuclear cell component in experimental immunological glomerulonephritis.
J Exp Med. 1978 Feb 1;147(2):369-84. doi: 10.1084/jem.147.2.369.
2
Role of the terminal complement pathway in experimental membranous nephropathy in the rabbit.
J Clin Invest. 1983 Dec;72(6):1948-57. doi: 10.1172/JCI111159.

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Intrarenal renin-angiotensin system activation and macrophage infiltrations in pediatric chronic glomerulonephritis.
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Patrolling monocytes promote intravascular neutrophil activation and glomerular injury in the acutely inflamed glomerulus.
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Voltage-gated potassium channel Kv1.3 blocker as a potential treatment for rat anti-glomerular basement membrane glomerulonephritis.
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Adenosine A2A receptor activation and macrophage-mediated experimental glomerulonephritis.
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A ROLE OF POLYMORPHONUCLEAR LEUKOCYTES AND COMPLEMENT IN NEPHROTOXIC NEPHRITIS.
J Exp Med. 1965 Jul 1;122(1):99-116. doi: 10.1084/jem.122.1.99.
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THE ORIGIN OF MACROPHAGES FROM BONE MARROW IN THE RAT.
Br J Exp Pathol. 1965 Feb;46(1):62-70.
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EXPERIMENTAL GLOMERULONEPHRITIS. VI. THE AUTOLOGOUS PHASE OF NEPHROTOXIC SERUM NEPHRITIS.
J Exp Med. 1965 May 1;121(5):715-25. doi: 10.1084/jem.121.5.715.
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EXPERIMENTAL GLOMERULONEPHRITIS. IV. PARTICIPATION OF COMPLEMENT IN NEPHROTOXIC NEPHRITIS.
J Exp Med. 1964 Jan 1;119(6):965-82. doi: 10.1084/jem.119.6.965.
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The origin and kinetics of mononuclear phagocytes.
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The pathogenesis of glomerulonephritis.
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