[脑线粒体中毒性和创伤性水肿肿胀时氧化磷酸化的状态]
[Status of oxidative phosphorylation in brain mitochondria during its toxic and traumatic edema-swelling].
作者信息
Sharov A N, Novikov V E
出版信息
Vopr Med Khim. 1992 Sep-Oct;38(5):24-6.
PMID:1492390
Abstract
A rate of oxidative reactions was decreased in brain mitochondria under conditions of toxic edema and within 1 day after traumatic impairment of brain tissue. Oxidative phosphorylation in mitochondria was normalized within 4 days after trauma. These alterations appear to depend on inhibition of oxidative enzymes in the respiration chain.
摘要
在中毒性水肿情况下以及脑组织创伤损伤后1天内，脑线粒体中的氧化反应速率降低。创伤后4天内线粒体中的氧化磷酸化恢复正常。这些改变似乎取决于呼吸链中氧化酶的抑制作用。
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