Thurau S R
Augenklinik, Ludwig-Maximilians-Universität, München.
Ophthalmologe. 2005 May;102(5):485-90. doi: 10.1007/s00347-005-1183-9.
Cystoid macular edema (CME) is the most frequent cause of visual deterioration in uveitis patients. Intraocular inflammation disturbs the blood-retina barrier and leads to retinal edema. The basis of successful treatment is the anti-inflammatory and immunosuppressive therapy of uveitis. Restoration of the blood-retina barrier is mediated by corticosteroids and nonsteroidal anti-inflammatory agents. Resorption of extracellular fluid is improved by systemic carboanhydrase inhibitors. Despite aggressive therapy loss of visual acuity is frequent. Therefore, early diagnosis of CME and initiation of treatment, even if visual acuity is not yet impeded, is mandatory.
黄斑囊样水肿（CME）是葡萄膜炎患者视力下降最常见的原因。眼内炎症破坏血视网膜屏障，导致视网膜水肿。成功治疗的基础是对葡萄膜炎进行抗炎和免疫抑制治疗。血视网膜屏障的恢复由皮质类固醇和非甾体类抗炎药介导。全身性碳酸酐酶抑制剂可促进细胞外液的吸收。尽管进行了积极治疗，但视力丧失仍很常见。因此，即使视力尚未受到影响，对CME进行早期诊断并开始治疗也是必不可少的。
