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[心包镜检查——炎症性心脏病中心脏的新窗口]

[Pericardioscopy--a new window to the heart in inflammatory heart diseases].

作者信息

Maisch B, Drude L

机构信息

Abteilung für Innere Medizin, Schwerpunkt Kardiologie der Philipps-Universität Marburg.

出版信息

Herz. 1992 Apr;17(2):71-8.

PMID:1577366
Abstract

Pericardioscopy is a new diagnostic tool to visualize macroscopically alterations of both the epicardium and pericardium. For the first time the macropathology of the epicarditis and pericarditis can be observed in vivo by the cardiologist in viral, bacterial, uremic and autoimmune cardiac processes. It enables us also to get an insight into neoplastic and metastatic processes affecting the heart and the pericardium. The prerequisite is the documentation of a larger pericardial (greater than 150 ml by the cubic model) effusion with an echocardiographically documented effusion of type C (Figure 1b), which must have at least 5 mm separation of the epicardial and pericardial layer in diastole at the anterior side of the heart when echocardiographic imaging is performed from the subxyphoidal or third intercostal space (Figure 1a). The first experience in 30 patients is reported. After puncture of the pericardial effusion a 9F sheath was introduced by use of a guide wire under echocardiographic and/or x-ray control (Figure 3). The fluid was removed by aspiration and 100 to 150 ml of body warm saline were infused in the pericardial sack. A flexible 8F fiber glass instrument (Vantec, Baxter or Storz) and a rigid 110 degrees 8F endoscope (Storz) were used for visualization of peri- and epicardium and for video documentation (Figure 2). After endoscopic inspection of the macropathology fibrinous strands (Figure 4) or increased vascular injection (Figure 5) can be observed in viral, autoimmune or idiopathic pericarditis or perimyocarditis. In the latter three forms of pericardial effusion only inflammatory cells (Figure 6) can be observed when the pericardial fluid is analyzed.(ABSTRACT TRUNCATED AT 250 WORDS)

摘要

心包镜检查是一种新的诊断工具,可宏观观察心外膜和心包的病变。心脏病专家首次能够在病毒、细菌、尿毒症和自身免疫性心脏疾病过程中,在体内观察到心内膜炎和心包炎的大体病理变化。它还使我们能够深入了解影响心脏和心包的肿瘤及转移过程。前提条件是通过超声心动图记录到较大的心包积液(根据立方模型大于150毫升),且为C型积液(图1b),当从剑突下或第三肋间间隙进行超声心动图成像时,心脏前侧舒张期的心外膜和心包层至少有5毫米的分离(图1a)。报告了30例患者的首次经验。在心包穿刺抽液后,在超声心动图和/或X线引导下,通过导丝插入一个9F鞘管(图3)。抽出积液后,向心包腔内注入100至150毫升体温的生理盐水。使用一根柔性8F玻璃纤维器械(Vantec、百特或史托斯)和一根刚性110度8F内窥镜(史托斯)来观察心包和心外膜,并进行视频记录(图2)。在内窥镜检查大体病理时,在病毒、自身免疫或特发性心包炎或心包心肌炎中可观察到纤维蛋白条索(图4)或血管充血增加(图5)。在后三种形式的心包积液中,分析心包液时仅能观察到炎症细胞(图6)。(摘要截选至250词)

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