填补复制重启途径中的空白。
Filling the gaps in replication restart pathways.
作者信息
Lovett Susan T
机构信息
Rosenstiel Basic Medical Sciences Research Center, Department of Biology, Brandeis University, Waltham, MA 02454, USA.
出版信息
Mol Cell. 2005 Mar 18;17(6):751-2. doi: 10.1016/j.molcel.2005.03.001.
PMID:15780929
Abstract
Recent work by clarifies two mechanisms of replication fork assembly in E. coli: in vitro, the presence of a leading nascent strand dictates a PriA/B DnaT-dependent mechanism, whereas a gapped fork specifies PriC-mediated restart.
摘要
近期的研究阐明了大肠杆菌中复制叉组装的两种机制
在体外，前导新生链的存在决定了一种依赖PriA/B DnaT的机制，而有缺口的复制叉则指定了由PriC介导的重新启动。
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