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引用本文的文献

1
Perforated peptic ulcer.穿孔性消化性溃疡
Lancet. 2015 Sep 26;386(10000):1288-1298. doi: 10.1016/S0140-6736(15)00276-7.

本文引用的文献

1
IL1RN polymorphic gene and cagA-positive status independently increase the risk of noncardia gastric carcinoma.白细胞介素1受体拮抗剂多态性基因和细胞毒素相关基因A阳性状态独立增加非贲门胃癌的风险。
Int J Cancer. 2005 Jul 10;115(5):678-83. doi: 10.1002/ijc.20935.
2
IL1B and IL1RN polymorphic genes and Helicobacter pylori cagA strains decrease the risk of reflux esophagitis.白细胞介素1β(IL1B)和白细胞介素1受体拮抗剂(IL1RN)多态性基因以及幽门螺杆菌细胞毒素相关基因A(cagA)菌株可降低反流性食管炎的风险。
Gastroenterology. 2004 Jul;127(1):73-9. doi: 10.1053/j.gastro.2004.03.069.
3
Association of interleukin 1 gene family polymorphisms with duodenal ulcer disease.白细胞介素1基因家族多态性与十二指肠溃疡病的关联
Clin Exp Immunol. 2003 Dec;134(3):525-31. doi: 10.1111/j.1365-2249.2003.02325.x.
4
A proinflammatory genetic profile increases the risk for chronic atrophic gastritis and gastric carcinoma.促炎基因谱增加慢性萎缩性胃炎和胃癌的风险。
Gastroenterology. 2003 Aug;125(2):364-71. doi: 10.1016/s0016-5085(03)00899-0.
5
Increased risk of noncardia gastric cancer associated with proinflammatory cytokine gene polymorphisms.促炎细胞因子基因多态性与非贲门胃癌风险增加相关。
Gastroenterology. 2003 May;124(5):1193-201. doi: 10.1016/s0016-5085(03)00157-4.
6
Interleukin-10 genotypes associate with the risk of gastric carcinoma in Taiwanese Chinese.白细胞介素-10基因分型与台湾华裔人群患胃癌的风险相关。
Int J Cancer. 2003 May 1;104(5):617-23. doi: 10.1002/ijc.10987.
7
Helicobacter pylori virulence genes and host IL-1RN and IL-1beta genes interplay in favouring the development of peptic ulcer and intestinal metaplasia.幽门螺杆菌毒力基因与宿主白细胞介素-1受体拮抗剂(IL-1RN)基因和白细胞介素-1β(IL-1β)基因相互作用,促进消化性溃疡和肠化生的发展。
Cytokine. 2002 Jun 7;18(5):242-51. doi: 10.1006/cyto.2002.0891.
8
Interleukin 1beta polymorphisms increase risk of hypochlorhydria and atrophic gastritis and reduce risk of duodenal ulcer recurrence in Japan.在日本,白细胞介素1β基因多态性会增加胃酸过少和萎缩性胃炎的风险,并降低十二指肠溃疡复发的风险。
Gastroenterology. 2002 Jul;123(1):92-105. doi: 10.1053/gast.2002.34156.
9
Density of Helicobacter pylori infection in patients with peptic ulcer perforation.消化性溃疡穿孔患者幽门螺杆菌感染密度
J Am Coll Surg. 1998 Jun;186(6):659-63. doi: 10.1016/s1072-7515(98)00128-8.
10
Role of Helicobacter pylori infection in perforation of peptic ulcer: an age- and gender-matched case-control study.幽门螺杆菌感染在消化性溃疡穿孔中的作用:一项年龄和性别匹配的病例对照研究。
J Clin Gastroenterol. 1997;25 Suppl 1:S235-9. doi: 10.1097/00004836-199700001-00037.

IL-1 gene cluster and TNFA-307 polymorphisms in the risk of perforated duodenal ulcer.

作者信息

Guerra J B, Rocha G A, Rocha A M C, de Castro Mendes C M, Saraiva I E B, de Oliveira C A, Queiroz D M M

出版信息

Gut. 2006 Jan;55(1):132-3. doi: 10.1136/gut.2005.077362.

DOI:10.1136/gut.2005.077362
PMID:16344580
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1856378/
Abstract
摘要