免疫血清对感染布鲁氏菌的巨噬细胞的介质依赖性活性。
Medium-dependent activity of immune serum on Brucella-infected macrophages.
机构信息
Division of Medical Microbiology and Immunology, School of Public Health, University of California, Berkeley, California 94720.
出版信息
Infect Immun. 1971 Feb;3(2):361-2. doi: 10.1128/iai.3.2.361-362.1971.
Abstract
Medium constituents affect the inhibitory action of anti-Brucella melitensis serum during in vitro infection of oil-induced peritoneal macrophages of the guinea pig with B. melitensis.
摘要
中等成分影响抗布鲁氏菌 melitensis 血清在布鲁氏菌 melitensis 感染诱导的豚鼠腹腔巨噬细胞体外感染过程中的抑制作用。
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本文引用的文献
1
Response of peritoneal exudate cells to Brucella melitensis. Influence of nature ox inflammatory irritant.
Proc Soc Exp Biol Med. 1960 Jun;104:295-7. doi: 10.3181/00379727-104-25814.
2
A cellular basis of immunity in experimental Brucella infection.
J Exp Med. 1958 Sep 1;108(3):343-60. doi: 10.1084/jem.108.3.343.
3
Phagocytosis and intracellular fate of Pasteurella tularensis: in vitro effects of exudate stimulants and streptomycin on phagocytic cells.
J Reticuloendothel Soc. 1967 Jan;4(1):10-23.
4
Serum-mediated immune cellular responses to Brucella melitensis REV 1. II. Restriction of Brucella by immune sera and macrophages.
J Reticuloendothel Soc. 1969 Apr;6(2):109-39.
5
Modification of macrophage function.
J Reticuloendothel Soc. 1968 Jun;5(3):179-202.
6
The host response to Calmette-Guérin bacillus infection in mice.
J Exp Med. 1969 May 1;129(5):1079-107. doi: 10.1084/jem.129.5.1079.
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