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Molecular injury to mitochondria produced by ethanol and acetaldehyde.

作者信息

Cederbaum A I, Rubin E

出版信息

Fed Proc. 1975 Oct;34(11):2045-51.

PMID:170139
Abstract
摘要

相似文献

1
Molecular injury to mitochondria produced by ethanol and acetaldehyde.乙醇和乙醛对线粒体造成的分子损伤。
Fed Proc. 1975 Oct;34(11):2045-51.
2
Effect of acetaldehyde on activity of shuttles for the transport of reducing equivalents into the mitochondria.乙醛对还原当量转运至线粒体的穿梭系统活性的影响。
FEBS Lett. 1973 Nov 15;37(1):89-92. doi: 10.1016/0014-5793(73)80432-6.
3
The mitochondrial respiratory control and oxidative phosphorylation of ethanol fed rats.
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4
Effects of clofibrate on mitochondrial function.
Biochem Pharmacol. 1974 Jul 15;23(14):1985-96. doi: 10.1016/0006-2952(74)90257-3.
5
Hydrogen transfer into mitochondria in the metabolism of ethanol. II. Effect of disulfiram on the hydrogen and energy transfer.
Ann Med Exp Biol Fenn. 1967;45(1):46-56.
6
Effects of pyrazole, 4-bromopyrazole and 4-methylpyrazole on mitochondrial function.
Biochem Pharmacol. 1974 Jan 15;23(2):203-13. doi: 10.1016/0006-2952(74)90411-0.
7
Effects of alcohol on hepatic metabolism.酒精对肝脏代谢的影响。
Rev Eur Etud Clin Biol. 1971 Dec;16(10):965-9.
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Consequences dietary ethanol on permeability and respiration of mitochondria and liver ADH in young and aged rats.膳食乙醇对年轻和老年大鼠线粒体通透性、呼吸及肝脏乙醇脱氢酶的影响。
Curr Alcohol. 1979;5:91-100.
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Effect of chronic ethanol consumption and acetaldehyde on partial reactions of oxidative phosphorylation and CO2 production from citric acid cycle intermediates.长期乙醇摄入及乙醛对氧化磷酸化部分反应和柠檬酸循环中间产物二氧化碳生成的影响。
Arch Biochem Biophys. 1976 Oct;176(2):525-38. doi: 10.1016/0003-9861(76)90196-x.
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Metabolic consequences of chronic alcohol consumption: attenuation of hepatic redox changes despite enhanced capacity to eliminate ethanol.长期饮酒的代谢后果:尽管消除乙醇的能力增强,但肝脏氧化还原变化减弱。
Curr Alcohol. 1979;5:109-18.

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Ethanol-induced oxidative stress: basic knowledge.乙醇诱导的氧化应激:基础知识。
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Hepatic steatosis in response to acute alcohol exposure in zebrafish requires sterol regulatory element binding protein activation.斑马鱼急性酒精暴露后发生的肝脏脂肪变性需要固醇调节元件结合蛋白激活。
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Oxidant-induced atrogin-1 and transforming growth factor-beta1 precede alcohol-related myopathy in rats.氧化应激诱导的肌萎缩蛋白-1和转化生长因子-β1在大鼠酒精相关性肌病之前出现。
Muscle Nerve. 2007 Dec;36(6):842-8. doi: 10.1002/mus.20883.
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Effect of chronic ethanol feeding on glutathione and functional integrity of mitochondria in periportal and perivenous rat hepatocytes.长期乙醇喂养对大鼠肝门周和肝静脉周肝细胞中谷胱甘肽及线粒体功能完整性的影响。
J Clin Invest. 1994 Jul;94(1):193-201. doi: 10.1172/JCI117306.
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Pathobiochemical transition of secondary coproporphyrinuria to chronic hepatic porphyria in humans.人类继发性粪卟啉尿症向慢性肝卟啉症的病理生化转变。
Klin Wochenschr. 1980 Feb 1;58(3):141-8. doi: 10.1007/BF01477271.
7
Ultrastructural morphometric study on the rat heart after chronic ethanol feeding.慢性乙醇喂养后大鼠心脏的超微结构形态计量学研究。
Virchows Arch A Pathol Anat Histol. 1980;389(1):59-77. doi: 10.1007/BF00428668.
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The effect of methylene blue on the hepatocellular redox state and liver lipid content during chronic ethanol feeding in the rat.亚甲蓝对大鼠慢性乙醇喂养期间肝细胞氧化还原状态和肝脏脂质含量的影响。
Biochem J. 1985 Dec 15;232(3):877-82. doi: 10.1042/bj2320877.
9
Alcohol and body weight in United States adults.美国成年人的酒精与体重
Am J Public Health. 1987 Oct;77(10):1324-30. doi: 10.2105/ajph.77.10.1324.
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Alcohol and acetaldehyde metabolism in Caucasians, Chinese and Amerinds.高加索人、中国人和美洲印第安人的酒精和乙醛代谢
Can Med Assoc J. 1976 Nov 6;115(9):851-5.