相似文献
1
SARS-CoV, but not HCoV-NL63, utilizes cathepsins to infect cells: viral entry.
Adv Exp Med Biol. 2006;581:335-8. doi: 10.1007/978-0-387-33012-9_60.
2
SARS coronavirus, but not human coronavirus NL63, utilizes cathepsin L to infect ACE2-expressing cells.
J Biol Chem. 2006 Feb 10;281(6):3198-203. doi: 10.1074/jbc.M508381200. Epub 2005 Dec 8.
3
Differential downregulation of ACE2 by the spike proteins of severe acute respiratory syndrome coronavirus and human coronavirus NL63.
J Virol. 2010 Jan;84(2):1198-205. doi: 10.1128/JVI.01248-09. Epub 2009 Oct 28.
4
The S proteins of human coronavirus NL63 and severe acute respiratory syndrome coronavirus bind overlapping regions of ACE2.
Virology. 2007 Oct 25;367(2):367-74. doi: 10.1016/j.virol.2007.04.035. Epub 2007 Jul 12.
5
Human coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry.
Proc Natl Acad Sci U S A. 2005 May 31;102(22):7988-93. doi: 10.1073/pnas.0409465102. Epub 2005 May 16.
6
Highly conserved regions within the spike proteins of human coronaviruses 229E and NL63 determine recognition of their respective cellular receptors.
J Virol. 2006 Sep;80(17):8639-52. doi: 10.1128/JVI.00560-06.
7
Inhibition of endoplasmic reticulum-resident glucosidases impairs severe acute respiratory syndrome coronavirus and human coronavirus NL63 spike protein-mediated entry by altering the glycan processing of angiotensin I-converting enzyme 2.
Antimicrob Agents Chemother. 2015 Jan;59(1):206-16. doi: 10.1128/AAC.03999-14. Epub 2014 Oct 27.
8
Proteolysis of SARS-associated coronavirus spike glycoprotein.
Adv Exp Med Biol. 2006;581:235-40. doi: 10.1007/978-0-387-33012-9_39.
9
TACE antagonists blocking ACE2 shedding caused by the spike protein of SARS-CoV are candidate antiviral compounds.
Antiviral Res. 2010 Mar;85(3):551-5. doi: 10.1016/j.antiviral.2009.12.001. Epub 2009 Dec 6.
10
Interaction between the spike protein of human coronavirus NL63 and its cellular receptor ACE2.
Adv Exp Med Biol. 2006;581:281-4. doi: 10.1007/978-0-387-33012-9_47.
引用本文的文献
1
Genome-scale CRISPR screen identifies TMEM41B as a multi-function host factor required for coronavirus replication.
PLoS Pathog. 2021 Dec 6;17(12):e1010113. doi: 10.1371/journal.ppat.1010113. eCollection 2021 Dec.
2
An Engineered Receptor-Binding Domain Improves the Immunogenicity of Multivalent SARS-CoV-2 Vaccines.
mBio. 2021 May 11;12(3):e00930-21. doi: 10.1128/mBio.00930-21.
3
Single-cell multiomic profiling of human lungs reveals cell-type-specific and age-dynamic control of SARS-CoV2 host genes.
Elife. 2020 Nov 9;9:e62522. doi: 10.7554/eLife.62522.
4
The role of cysteine peptidases in coronavirus cell entry and replication: The therapeutic potential of cathepsin inhibitors.
PLoS Pathog. 2020 Nov 2;16(11):e1009013. doi: 10.1371/journal.ppat.1009013. eCollection 2020 Nov.
5
Targeting the sAC-Dependent cAMP Pool to Prevent SARS-Cov-2 Infection.
Cells. 2020 Aug 25;9(9):1962. doi: 10.3390/cells9091962.
6
Structural and functional properties of SARS-CoV-2 spike protein: potential antivirus drug development for COVID-19.
Acta Pharmacol Sin. 2020 Sep;41(9):1141-1149. doi: 10.1038/s41401-020-0485-4. Epub 2020 Aug 3.
7
Viral-Induced Enhanced Disease Illness.
Front Microbiol. 2018 Dec 5;9:2991. doi: 10.3389/fmicb.2018.02991. eCollection 2018.
8
Host Factors in Coronavirus Replication.
Curr Top Microbiol Immunol. 2018;419:1-42. doi: 10.1007/82_2017_25.
9
Inhibition of endoplasmic reticulum-resident glucosidases impairs severe acute respiratory syndrome coronavirus and human coronavirus NL63 spike protein-mediated entry by altering the glycan processing of angiotensin I-converting enzyme 2.
Antimicrob Agents Chemother. 2015 Jan;59(1):206-16. doi: 10.1128/AAC.03999-14. Epub 2014 Oct 27.
10
The pathology and pathogenesis of experimental severe acute respiratory syndrome and influenza in animal models.
J Comp Pathol. 2014 Jul;151(1):83-112. doi: 10.1016/j.jcpa.2014.01.004. Epub 2014 Jan 15.
本文引用的文献
1
Human coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry.
Proc Natl Acad Sci U S A. 2005 May 31;102(22):7988-93. doi: 10.1073/pnas.0409465102. Epub 2005 May 16.
2
Endosomal proteolysis of the Ebola virus glycoprotein is necessary for infection.
Science. 2005 Jun 10;308(5728):1643-5. doi: 10.1126/science.1110656. Epub 2005 Apr 14.
3
Virus entry: molecular mechanisms and biomedical applications.
Nat Rev Microbiol. 2004 Feb;2(2):109-22. doi: 10.1038/nrmicro817.
4
Characterization of severe acute respiratory syndrome-associated coronavirus (SARS-CoV) spike glycoprotein-mediated viral entry.
Proc Natl Acad Sci U S A. 2004 Mar 23;101(12):4240-5. doi: 10.1073/pnas.0306446101. Epub 2004 Mar 9.
5
Expression cloning of functional receptor used by SARS coronavirus.
Biochem Biophys Res Commun. 2004 Mar 5;315(2):439-44. doi: 10.1016/j.bbrc.2004.01.076.
6
A 193-amino acid fragment of the SARS coronavirus S protein efficiently binds angiotensin-converting enzyme 2.
J Biol Chem. 2004 Jan 30;279(5):3197-201. doi: 10.1074/jbc.C300520200. Epub 2003 Dec 11.
7
Angiotensin-converting enzyme 2 is a functional receptor for the SARS coronavirus.
Nature. 2003 Nov 27;426(6965):450-4. doi: 10.1038/nature02145.
8
Identification of a receptor-binding domain of the spike glycoprotein of human coronavirus HCoV-229E.
J Virol. 2003 Feb;77(4):2530-8. doi: 10.1128/jvi.77.4.2530-2538.2003.
9
Addition of exogenous protease facilitates reovirus infection in many restrictive cells.
J Virol. 2002 Aug;76(15):7430-43. doi: 10.1128/jvi.76.15.7430-7443.2002.
10
Cathepsin L and cathepsin B mediate reovirus disassembly in murine fibroblast cells.
J Biol Chem. 2002 Jul 5;277(27):24609-17. doi: 10.1074/jbc.M201107200. Epub 2002 May 1.
Zotero 插件