Capone A, Meredith T A
Department of Ophthalmology, Emory University, Atlanta, Georgia.
Retina. 1990;10(4):265-8. doi: 10.1097/00006982-199010000-00007.
The authors report a 65-year-old healthy, white man who experienced a dramatic loss of central vision. Iris neovascularization, rubeotic glaucoma, disc neovascularization and subhyaloid hemorrhage developed after multiple, recurrent, idiopathic branch retinal arterial occlusions. Vitreous and perivascular inflammation were prominent associated clinical features. Systemic steroids were useful in suppressing intraocular and perivascular inflammation, yet neither steroid nor anticoagulant therapy effectively prevented recurrent occlusive episodes. Retinal neovascularization and rubeotic glaucoma were successfully managed with scatter panretinal photocoagulation. Episodic intraocular inflammation and ocular neovascularization have been noted in one-third of patients sustaining recurrent idiopathic branch retinal arterial occlusions.
作者报告了一名65岁健康的白人男性，其经历了严重的中心视力丧失。在多次复发性特发性视网膜分支动脉阻塞后，出现了虹膜新生血管、新生血管性青光眼、视盘新生血管和玻璃膜下出血。玻璃体和血管周围炎症是突出的相关临床特征。全身用类固醇有助于抑制眼内和血管周围炎症，但类固醇和抗凝治疗均不能有效预防复发性阻塞发作。视网膜新生血管和新生血管性青光眼通过全视网膜光凝成功得到控制。在三分之一患有复发性特发性视网膜分支动脉阻塞的患者中，已观察到发作性眼内炎症和眼新生血管。
