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Is ranolazine an antiarrhythmic drug?

作者信息

Eckhardt Lee L, Teelin Tom C, January Craig T

机构信息

Division of Cardiovascular Medicine, University of Wisconsin, Madison, WI 53792, USA.

出版信息

Am J Physiol Heart Circ Physiol. 2008 May;294(5):H1989-91. doi: 10.1152/ajpheart.00285.2008. Epub 2008 Mar 28.

DOI:10.1152/ajpheart.00285.2008
PMID:18375723
Abstract
摘要

相似文献

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Is ranolazine an antiarrhythmic drug?雷诺嗪是一种抗心律失常药物吗?
Am J Physiol Heart Circ Physiol. 2008 May;294(5):H1989-91. doi: 10.1152/ajpheart.00285.2008. Epub 2008 Mar 28.
2
An increase of late sodium current induces delayed afterdepolarizations and sustained triggered activity in atrial myocytes.晚钠电流增加会在心房肌细胞中诱发延迟后去极化和持续性触发活动。
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Molecular basis of ranolazine block of LQT-3 mutant sodium channels: evidence for site of action.雷诺嗪对LQT-3突变钠通道的阻滞作用的分子基础:作用位点的证据
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Potential application of late sodium current blockade in the treatment of heart failure and atrial fibrillation.晚期钠电流阻断在心力衰竭和心房颤动治疗中的潜在应用。
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Atrium-selective sodium channel block as a strategy for suppression of atrial fibrillation: differences in sodium channel inactivation between atria and ventricles and the role of ranolazine.心房选择性钠通道阻滞作为抑制心房颤动的策略:心房与心室钠通道失活的差异及雷诺嗪的作用
Circulation. 2007 Sep 25;116(13):1449-57. doi: 10.1161/CIRCULATIONAHA.107.704890. Epub 2007 Sep 4.
6
Ranolazine and late cardiac sodium current--a therapeutic target for angina, arrhythmia and more?雷诺嗪与晚期心脏钠电流——心绞痛、心律失常及其他疾病的治疗靶点?
Br J Pharmacol. 2006 May;148(1):4-6. doi: 10.1038/sj.bjp.0706713.
7
A slowly inactivating sodium current contributes to spontaneous diastolic depolarization of atrial myocytes.一种缓慢失活的钠电流有助于心房肌细胞的自发性舒张期去极化。
Am J Physiol Heart Circ Physiol. 2009 Oct;297(4):H1254-62. doi: 10.1152/ajpheart.00444.2009. Epub 2009 Aug 21.
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Enhanced late na(+) currents in atrial fibrillation new drug target or just an epiphenomenon?心房颤动中增强的晚期钠电流:新的药物靶点还是仅仅是一种附带现象?
J Am Coll Cardiol. 2010 May 25;55(21):2343-5. doi: 10.1016/j.jacc.2010.01.046.
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A focus on antiarrhythmic properties of ranolazine.专注于雷诺嗪的抗心律失常特性。
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Ranolazine for congenital and acquired late INa-linked arrhythmias: in silico pharmacological screening.雷诺嗪治疗先天性和获得性晚期 INa 相关心律失常:计算机药理学筛选。
Circ Res. 2013 Sep 13;113(7):e50-e61. doi: 10.1161/CIRCRESAHA.113.301971. Epub 2013 Jul 29.

引用本文的文献

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Tolterodine reduces veratridine-augmented late I, reverse-I and early afterdepolarizations in isolated rabbit ventricular myocytes.托特罗定可减少离体兔心室肌细胞中藜芦碱增强的晚期 I 、逆向 I 和早期后去极化。
Acta Pharmacol Sin. 2016 Nov;37(11):1432-1441. doi: 10.1038/aps.2016.76. Epub 2016 Aug 29.
2
Revisiting the ionic mechanisms of early afterdepolarizations in cardiomyocytes: predominant by Ca waves or Ca currents?重新探讨心肌细胞早期后除极的离子机制:主要由钙波还是钙电流引起?
Am J Physiol Heart Circ Physiol. 2012 Apr 15;302(8):H1636-44. doi: 10.1152/ajpheart.00742.2011. Epub 2012 Feb 3.
3
Diastolic transient inward current in long QT syndrome type 3 is caused by Ca2+ overload and inhibited by ranolazine.
3型长QT综合征中的舒张期短暂内向电流由Ca2+超载引起,并被雷诺嗪抑制。
J Mol Cell Cardiol. 2009 Aug;47(2):326-34. doi: 10.1016/j.yjmcc.2009.04.003. Epub 2009 Apr 14.
4
Regulation of intracellular and mitochondrial sodium in health and disease.健康与疾病状态下细胞内及线粒体钠的调节
Circ Res. 2009 Feb 13;104(3):292-303. doi: 10.1161/CIRCRESAHA.108.189050.