Citro Rudolfo, Patella Marco Mariano, Bossone Eduardo, Maione AntonGiulio, Provenza Gennaro, Gregorio Giovanni
UO UTIC-Cardiologia San Luca Hospital, Vallo della Lucania SA, Italy.
J Cardiovasc Med (Hagerstown). 2008 May;9(5):501-5. doi: 10.2459/JCM.0b013e3282f03aca.
We report a case of transient tako-tsubo cardiomyopathy characterized by an unusual trigger in a woman victim of near-drowning syndrome. After 24 h, electrocardiogram changes and a typical echocardiographic pattern of apical ballooning with a mild increase of serum troponin level induced the suspicion of tako-tsubo cardiomyopathy despite the absence of chest pain. Left ventriculography confirmed the apical ballooning, and coronary angiography revealed normal coronary arteries. Electrocardiogram changes and apical contraction abnormalities were reversed within 1 month. In conclusion, we hypothesize that hypoxemia related to near-drowning syndrome could have induced transient myocardial dysfunction mediated by a sympathetic nerve activation.
我们报告一例短暂性应激性心肌病,其特征为一名近乎溺水综合征女性受害者出现异常诱因。24小时后,尽管无胸痛症状,但心电图改变以及典型的心尖部气球样变超声心动图表现,同时血清肌钙蛋白水平轻度升高,引发了对应激性心肌病的怀疑。左心室造影证实了心尖部气球样变,冠状动脉造影显示冠状动脉正常。心电图改变和心尖部收缩异常在1个月内恢复正常。总之,我们推测与近乎溺水综合征相关的低氧血症可能通过交感神经激活诱发了短暂性心肌功能障碍。