Inositol 1,4,5-trisphosphate 3-kinase activity in high-speed supernatants from rat pancreatoma cells, AR4-2J.
作者信息
Menniti F S, Putney J W
出版信息
Biochem J. 1991 Mar 1;274 ( Pt 2)(Pt 2):622-3. doi: 10.1042/bj2740622.
Abstract
摘要
相似文献
1
Inositol 1,4,5-trisphosphate 3-kinase activity in high-speed supernatants from rat pancreatoma cells, AR4-2J.大鼠胰腺癌细胞AR4-2J高速上清液中的肌醇1,4,5-三磷酸3-激酶活性
Biochem J. 1991 Mar 1;274 ( Pt 2)(Pt 2):622-3. doi: 10.1042/bj2740622.
2
Identification in extracts from AR4-2J cells of inositol 1,4,5-trisphosphate by its susceptibility to inositol 1,4,5-trisphosphate 3-kinase and 5-phosphatase.通过对肌醇1,4,5-三磷酸3-激酶和5-磷酸酶的敏感性在AR4-2J细胞提取物中鉴定肌醇1,4,5-三磷酸
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3
Protein kinase C phosphorylates the inositol 1,4,5-trisphosphate receptor type 2 and decreases the mobilization of Ca2+in pancreatoma AR4-2J cells.蛋白激酶C使2型肌醇1,4,5-三磷酸受体磷酸化，并减少胰腺癌细胞AR4-2J中Ca2+的动员。
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Relationship between the calcium-mobilizing action of inositol 1,4,5-trisphosphate in permeable AR4-2J cells and the estimated levels of inositol 1,4,5-trisphosphate in intact AR4-2J cells.可渗透的AR4-2J细胞中肌醇1,4,5-三磷酸的钙动员作用与完整AR4-2J细胞中肌醇1,4,5-三磷酸估计水平之间的关系。
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The inositol 1,4,5-trisphosphate 6-kinase of Schizosaccharomyces pombe.粟酒裂殖酵母的肌醇1,4,5-三磷酸6-激酶
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Ca2+ dependence of inositol 1,4,5-trisphosphate 3-kinase activity in the cytosol fraction of pig coronary artery.猪冠状动脉胞质溶胶部分中肌醇1,4,5-三磷酸3-激酶活性的钙离子依赖性
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Phosphorylation by protein kinase C inactivates an inositol 1,4,5-trisphosphate 3-kinase purified from human platelets.蛋白激酶C介导的磷酸化作用会使从人血小板中纯化出的肌醇-1,4,5-三磷酸3-激酶失活。
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Detection of inositol 1,4,5-trisphosphate kinase in retina. A direct demonstration of phosphorylation of inositol 1,4,5-trisphosphate by ATP.视网膜中肌醇-1,4,5-三磷酸激酶的检测。ATP对肌醇-1,4,5-三磷酸磷酸化的直接证明。
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Chemical transmission at the triad: InsP3?
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The perturbation, by aluminium, of receptor-generated calcium transients in hepatocytes is not due to effects of Ins(1,4,5)P3-stimulated Ca2+ release or Ins(1,4,5)P3 metabolism by the 5-phosphatase and 3-kinase.铝对肝细胞中受体产生的钙瞬变的干扰并非由于Ins(1,4,5)P3刺激的Ca2+释放或5-磷酸酶和3-激酶对Ins(1,4,5)P3代谢的影响。
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本文引用的文献
1
Separation of multiple isomers of inositol phosphates formed in GH3 cells.GH3细胞中形成的肌醇磷酸多种异构体的分离。
Biochem J. 1987 Mar 1;242(2):361-6. doi: 10.1042/bj2420361.
2
The inositol tris/tetrakisphosphate pathway--demonstration of Ins(1,4,5)P3 3-kinase activity in animal tissues.肌醇三/四磷酸途径——动物组织中肌醇-1,4,5-三磷酸3-激酶活性的证明
Nature. 1986;320(6063):631-4. doi: 10.1038/320631a0.
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Metabolism of the inositol phosphates produced upon receptor activation.受体激活后产生的肌醇磷酸的代谢。
Biochem J. 1989 Jun 1;260(2):313-24. doi: 10.1042/bj2600313.
4
Formation and metabolism of [3H]inositol phosphates in AR42J pancreatoma cells. Substance P-induced Ca2+ mobilization in the apparent absence of inositol 1,4,5-trisphosphate 3-kinase activity.[3H]肌醇磷酸酯在AR42J胰腺癌细胞中的形成与代谢。在明显缺乏肌醇1,4,5-三磷酸3-激酶活性的情况下，P物质诱导的Ca2+动员。
J Biol Chem. 1988 Oct 25;263(30):15297-303.
5
Functional substance P receptors on a rat pancreatic acinar cell line.大鼠胰腺腺泡细胞系上的功能性P物质受体
J Neurosci. 1985 Dec;5(12):3370-8. doi: 10.1523/JNEUROSCI.05-12-03370.1985.
6
Origins of myo-inositol tetrakisphosphates in agonist-stimulated rat pancreatoma cells. Stimulation by bombesin of myo-inositol 1,3,4,5,6-pentakisphosphate breakdown to myo-inositol 3,4,5,6-tetrakisphosphate.激动剂刺激的大鼠胰腺癌细胞中肌醇四磷酸的来源。蛙皮素刺激肌醇1,3,4,5,6 - 五磷酸分解为肌醇3,4,5,6 - 四磷酸。
J Biol Chem. 1990 Jul 5;265(19):11167-76.
7
Identification in extracts from AR4-2J cells of inositol 1,4,5-trisphosphate by its susceptibility to inositol 1,4,5-trisphosphate 3-kinase and 5-phosphatase.通过对肌醇1,4,5-三磷酸3-激酶和5-磷酸酶的敏感性在AR4-2J细胞提取物中鉴定肌醇1,4,5-三磷酸
Biochem J. 1990 Jul 1;269(1):195-200. doi: 10.1042/bj2690195.
8
Transplantation of azaserine-induced carcinomas of pancreas in rats.大鼠中氮杂丝氨酸诱导的胰腺癌移植
Cancer Lett. 1979 Aug;7(4):197-202. doi: 10.1016/s0304-3835(79)80080-4.
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