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1
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Expert Rev Anti Infect Ther. 2008 Apr;6(2):201-8. doi: 10.1586/14787210.6.2.201.
2
An integrated model of the recognition of Candida albicans by the innate immune system.
Nat Rev Microbiol. 2008 Jan;6(1):67-78. doi: 10.1038/nrmicro1815.
3
Immune recognition of Candida albicans beta-glucan by dectin-1.
J Infect Dis. 2007 Nov 15;196(10):1565-71. doi: 10.1086/523110. Epub 2007 Oct 31.
4
Human epithelial cells establish direct antifungal defense through TLR4-mediated signaling.
J Clin Invest. 2007 Dec;117(12):3664-72. doi: 10.1172/JCI28115.
5
Functional yet balanced reactivity to Candida albicans requires TRIF, MyD88, and IDO-dependent inhibition of Rorc.
J Immunol. 2007 Nov 1;179(9):5999-6008. doi: 10.4049/jimmunol.179.9.5999.
6
IL-23 and the Th17 pathway promote inflammation and impair antifungal immune resistance.
Eur J Immunol. 2007 Oct;37(10):2695-706. doi: 10.1002/eji.200737409.
7
Monocyte responses to Candida albicans are enhanced by antibody in cooperation with antibody-independent pathogen recognition.
FEMS Immunol Med Microbiol. 2007 Oct;51(1):70-83. doi: 10.1111/j.1574-695X.2007.00278.x. Epub 2007 Jul 4.
8
Oral epithelium-Candida glabrata interactions in vitro.
Oral Microbiol Immunol. 2007 Jun;22(3):182-7. doi: 10.1111/j.1399-302X.2007.00342.x.
9
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10
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