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1
Rethinking T cell immunity in oropharyngeal candidiasis.
J Exp Med. 2009 Feb 16;206(2):269-73. doi: 10.1084/jem.20090093. Epub 2009 Feb 9.
2
Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis.
J Exp Med. 2009 Feb 16;206(2):299-311. doi: 10.1084/jem.20081463. Epub 2009 Feb 9.
3
Immunity to Candida.
Oral Dis. 2002;8 Suppl 2:69-75. doi: 10.1034/j.1601-0825.2002.00015.x.
4
Candida-host interactions in HIV disease: relationships in oropharyngeal candidiasis.
Adv Dent Res. 2006 Apr 1;19(1):80-4. doi: 10.1177/154407370601900116.
6
Candida-host interactions in HIV disease: implications for oropharyngeal candidiasis.
Adv Dent Res. 2011 Apr;23(1):45-9. doi: 10.1177/0022034511399284.
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Th1/Th2 cytokine expression in saliva of HIV-positive and HIV-negative individuals: a pilot study in HIV-positive individuals with oropharyngeal candidiasis.
J Acquir Immune Defic Syndr Hum Retrovirol. 1998 Dec 1;19(4):373-80. doi: 10.1097/00042560-199812010-00008.
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Host factors associated with HIV-related oral candidiasis. A review.
Oral Surg Oral Med Oral Pathol. 1992 Feb;73(2):181-6. doi: 10.1016/0030-4220(92)90192-s.
9
Cytokines in the oral mucosa of mice infected with Candida albicans.
Oral Microbiol Immunol. 2002 Dec;17(6):375-8. doi: 10.1034/j.1399-302x.2002.170607.x.

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1
Methods Related to the Immunopathogenesis of Vulvovaginal Candidiasis and Associated Neutrophil Anergy.
Methods Mol Biol. 2022;2542:193-218. doi: 10.1007/978-1-0716-2549-1_14.
2
The Role of IL-17 During Infections in the Female Reproductive Tract.
Front Immunol. 2022 Apr 14;13:861444. doi: 10.3389/fimmu.2022.861444. eCollection 2022.
4
Mucosal IgA Prevents Commensal Dysbiosis in the Oral Cavity.
Front Immunol. 2020 Oct 22;11:555363. doi: 10.3389/fimmu.2020.555363. eCollection 2020.
6
Human inborn errors of immunity underlying superficial or invasive candidiasis.
Hum Genet. 2020 Jun;139(6-7):1011-1022. doi: 10.1007/s00439-020-02141-7. Epub 2020 Mar 2.
7
Oral Candidiasis: A Disease of Opportunity.
J Fungi (Basel). 2020 Jan 16;6(1):15. doi: 10.3390/jof6010015.
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Candida albicans Pathogenesis: Fitting within the Host-Microbe Damage Response Framework.
Infect Immun. 2016 Sep 19;84(10):2724-39. doi: 10.1128/IAI.00469-16. Print 2016 Oct.

本文引用的文献

1
Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis.
J Exp Med. 2009 Feb 16;206(2):299-311. doi: 10.1084/jem.20081463. Epub 2009 Feb 9.
3
Differential Th17 CD4 T-cell depletion in pathogenic and nonpathogenic lentiviral infections.
Blood. 2008 Oct 1;112(7):2826-35. doi: 10.1182/blood-2008-05-159301. Epub 2008 Jul 29.
4
Mutations in STAT3 and IL12RB1 impair the development of human IL-17-producing T cells.
J Exp Med. 2008 Jul 7;205(7):1543-50. doi: 10.1084/jem.20080321.
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Accidental virulence, cryptic pathogenesis, martians, lost hosts, and the pathogenicity of environmental microbes.
Eukaryot Cell. 2007 Dec;6(12):2169-74. doi: 10.1128/EC.00308-07. Epub 2007 Oct 19.
10
IL-23 and the Th17 pathway promote inflammation and impair antifungal immune resistance.
Eur J Immunol. 2007 Oct;37(10):2695-706. doi: 10.1002/eji.200737409.

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