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对来自法国阿尔萨斯地区的 102 名常染色体隐性进行性小脑共济失调患者的流行病学、临床、辅助检查和分子研究:对临床管理的影响。

Epidemiological, clinical, paraclinical and molecular study of a cohort of 102 patients affected with autosomal recessive progressive cerebellar ataxia from Alsace, Eastern France: implications for clinical management.

机构信息

Département de Neurologie, Hôpital Civil, Centre Hospitalier Universitaire de Strasbourg, 1, place de l'Hôpital, 67000, Strasbourg, France.

出版信息

Neurogenetics. 2010 Feb;11(1):1-12. doi: 10.1007/s10048-009-0196-y. Epub 2009 May 14.

DOI:10.1007/s10048-009-0196-y
PMID:19440741
Abstract

While Friedreich's ataxia (FRDA) and ataxia telangiectasia (AT) are known to be the two most frequent forms of autosomal recessive cerebellar ataxia (ARCA), knowledge on the other forms of ARCA has been obtained only recently, and they appear to be rarer. Little is known about the epidemiological features and the relative frequency of the ARCAs and only few data are available about the comparative features of ARCAs. We prospectively studied 102 suspected ARCA cases from Eastern France (including 95 from the Alsace region) between 2002 and 2008. The diagnostic procedure was based on a sequential strategic scheme. We examined the clinical, paraclinical and molecular features of the large cohort of patients and compared features and epidemiology according to molecular diagnosis. A molecular diagnosis could be established for 57 patients; 36 were affected with FRDA, seven with ataxia plus oculomotor apraxia type 2 (AOA2), four with AT, three with ataxia plus oculomotor apraxia type 1 (AOA1), three with Marinesco-Sjögren syndrome, two with autosomal recessive spastic ataxia of Charlevoix-Saguenay (ARSACS), one with ataxia with vitamin E deficiency (AVED) and one with autosomal recessive cerebellar ataxia type 2 (ARCA2). The group of patients with no identified mutation had a significantly lower spinocerebellar degeneration functional score corrected for disease duration (SDFS/DD ratio; p = 0.002) and comprised a significantly higher proportion of cases with onset after 20 years (p < 0.01). Extensor plantar reflexes were rarer and cerebellar atrophy was more frequent in the group of patients with a known non-Friedreich ARCA compared to all other patients (p < 0.0001 and p = 0.0003, respectively). Lower limb areflexia and electroneuromyographic evidences of peripheral neuropathy were more frequent in the Friedreich ataxia group than in the group with a known non-Friedreich ataxia and were more frequent in the later group than in the group with no identified mutation (p = 0.0001 and p = 0.01, respectively). The overall prevalence of ARCA in Alsace is 1/19,000. We can infer the prevalence of FRDA in Alsace to be 1/50,000 and infer that AT is approximately eight times less frequent than FRDA. MSS, AOA2 and ARSACS appear only slightly less frequent than AT. Despite the broad variability of severity, Friedreich ataxia patients are clinically distinct from the other forms of ARCA. Patients with no identified mutation have more often a pure cerebellar degenerative disease or a spastic ataxia phenotype. It appears that ARCA cases can be divided into two major groups of different prognosis, an early-onset group with a highly probable genetic cause and an adult-onset group with better prognosis for which a genetic cause is more difficult to prove but not excluded. ARCAs are rare, early-disabling and genetically heterogeneous diseases dominated by FRDA. Several of the recently identified ARCAs, such as AVED, ARSACS, AOA1, AOA2 and MSS, have a prevalence close to AT and should be searched for extensively irrespective of ethnic origins. The strategic scheme is a useful tool for the diagnosis of ARCAs in clinical practice.

摘要

尽管弗里德里希共济失调(FRDA)和毛细血管扩张性共济失调(AT)是已知的两种最常见的常染色体隐性小脑共济失调(ARCA)形式,但最近才获得了其他形式的 ARCA 的知识,而且它们似乎更为罕见。关于 ARCA 的流行病学特征和相对频率知之甚少,只有少数关于 ARCA 比较特征的数据。我们前瞻性研究了 2002 年至 2008 年间法国东部(包括阿尔萨斯地区的 95 例)的 102 例疑似 ARCA 病例。诊断程序基于序贯战略方案。我们检查了大量患者的临床、临床前和分子特征,并根据分子诊断比较了特征和流行病学。57 例患者可明确分子诊断;36 例为 FRDA 患者,7 例为伴动眼运动不能 2 型(AOA2)的共济失调患者,4 例为 AT 患者,3 例为伴动眼运动不能 1 型(AOA1)的共济失调患者,3 例为 Marinesco-Sjögren 综合征患者,2 例为 Charlevoix-Saguenay 遗传性痉挛性共济失调(ARSACS)患者,1 例为维生素 E 缺乏性共济失调(AVED)患者,1 例为常染色体隐性小脑共济失调 2 型(ARCA2)患者。未发现突变的患者的脊髓小脑变性功能评分校正病程(SDFS/DD 比值;p = 0.002)显著较低,并且发病年龄在 20 岁以后的患者比例显著较高(p < 0.01)。与所有其他患者相比,已知非 FRDA 的 ARCA 患者的伸肌跖反射较少,小脑萎缩更为常见(p < 0.0001 和 p = 0.0003)。与具有已知非 FRDA 共济失调的患者相比,Friedreich 共济失调组的下肢反射消失和周围神经病的电神经图证据更为常见,与无基因突变的患者相比更为常见(p = 0.0001 和 p = 0.01)。阿尔萨斯的 ARCA 总体患病率为 1/19000。我们可以推断阿尔萨斯的 FRDA 患病率为 1/50000,并推断 AT 的频率大约比 FRDA 低 8 倍。MSS、AOA2 和 ARSACS 似乎比 AT 略低。尽管严重程度差异很大,但 Friedreich 共济失调患者在临床上与其他形式的 ARCA 不同。无基因突变的患者更常出现单纯性小脑退行性疾病或痉挛性共济失调表型。似乎 ARCA 病例可以分为预后不同的两个主要组,一个是早期发病组,遗传原因高度可能,另一个是成年发病组,预后较好,遗传原因更难证明但不能排除。ARCA 是罕见的、早期致残的、遗传异质性疾病,以 FRDA 为主。最近发现的几种 ARCA,如 AVED、ARSACS、AOA1、AOA2 和 MSS,其患病率与 AT 相近,无论种族来源如何,都应广泛寻找。该策略是临床实践中 ARCA 诊断的有用工具。

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