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弥漫性特发性骨肥厚症所致吞咽困难:五例分析。

Dysphagia due to diffuse idiopathic skeletal hyperostosis: an analysis of five cases.

机构信息

Department of Rehabilitation Medicine, University of Padua, Via Giustiniani 1, 35128, Padua, Italy.

出版信息

Rheumatol Int. 2010 Mar;30(5):681-5. doi: 10.1007/s00296-009-0967-7. Epub 2009 May 23.

Abstract

We report five cases of dysphagia caused by diffuse idiopathic skeletal hyperostosis or Forestier's disease of the cervical spine observed by clinical evaluations, X-ray and videofluoroscopy. Dysphagia in particular was present for solid foods in all patients. Three patients showed bolus aspiration into the airways, mainly in the post-swallowing phase and one patient with preserved epiglottic tilt showed post-swallowing penetration. The physiopathogenetic mechanisms affecting swallowing dynamics were mechanical compression of the pharyngeal lumen associated with abnormal epiglottic tilt, incomplete upper esophageal sphincter openings and epiglottic/vallecula stasis. According to our results we can hypothesize that these kinetic alterations may due to Forestier's disease.

摘要

我们报告了 5 例由弥漫性特发性骨肥厚或颈椎 Forestier 病引起的吞咽困难病例,这些病例通过临床评估、X 射线和荧光透视检查观察到。特别是所有患者都存在固体食物吞咽困难。3 名患者出现食团吸入气道,主要发生在吞咽后阶段,1 名具有保留杓状软骨倾斜的患者出现吞咽后穿透。影响吞咽动力学的病理生理机制是咽腔的机械性压迫与异常杓状软骨倾斜、不完全的食管上括约肌开放和杓状软骨/ vallecula 停滞有关。根据我们的结果,我们可以假设这些运动改变可能是由于 Forestier 病引起的。

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