辐射白血病病毒诱导肿瘤发生的遗传控制II. Srlv-1位点的影响，该位点与H-2不连锁
Genetic control of radiation leukemia virus-induced tumorigenesis II. Influence of Srlv-1, a locus not linked to H-2.
作者信息
Meruelo D, Lieberman M, Deak B, McDevitt H O
出版信息
J Exp Med. 1977 Oct 1;146(4):1088-95. doi: 10.1084/jem.146.4.1088.
Abstract
A single locus, tentatively denoted Srlv-1 (susceptibility to radiation leukemia virus [RadLV]-1), confers dominant susceptibility to RadLV-induced leukemogenesis. Srlv-1 is not linked to H-2, and appears to be distinct from Fv-1 and Fv-2. Preliminary data suggest that Srlv-1 affected virus proliferation. A striking feature of this system is that Srlv-1 overrides the protection afforded by the H2D-associated dominant resistance to RadLV-induced neoplasia.
摘要
一个暂定名为Srlv-1（对辐射白血病病毒[RadLV]-1易感）的单一基因座赋予对RadLV诱导白血病发生的显性易感性。Srlv-1与H-2不连锁，且似乎与Fv-1和Fv-2不同。初步数据表明Srlv-1影响病毒增殖。该系统的一个显著特征是Srlv-1克服了由H2D相关的对RadLV诱导肿瘤形成的显性抗性所提供的保护作用。
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