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1例Nd:YAG激光意外黄斑损伤及随后6年的随访

A case of accidental macular injury by Nd: YAG laser and subsequent 6 year follow-up.

作者信息

Park Dong Ho, Kim In Taek

机构信息

Department of Ophthalmology, School of Medicine, Kyungpook National University, Daegu, Korea.

出版信息

Korean J Ophthalmol. 2009 Sep;23(3):207-9. doi: 10.3341/kjo.2009.23.3.207. Epub 2009 Sep 8.

DOI:10.3341/kjo.2009.23.3.207
PMID:19794950
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2739961/
Abstract

Here, we report the case of a patient who sustained Nd: YAG laser macular injury with subsequent 6 year follow-up evaluation. A 23-year-old female was accidentally exposed to a Q-switched Nd: YAG laser without protective goggles. Upon initial evaluation, the best-corrected visual acuity of her affected eye was 20/100 OD. Fundoscopic examination revealed a macular laser burn and vitreous hemorrhage. Corticosteroids, in the form of 60 mg prednisolone, were administered orally with a 10 mg per week taper. Nineteen days following exposure, fundoscopic examination revealed a distinct epiretinal membrane which resolved within six months. The best-corrected visual acuity of the affected eye remained 20/100 OD. This clinical course is similar to those of previously reported cases including vitreous hemorrhage and subsequent epiretinal membrane formation. However, visual acuity did not recover despite spontaneous regression of the epiretinal membrane and at 6 year follow-up, there was neither choroidal neovascularization nor macular hole formation.

摘要

在此,我们报告一例患者,其遭受了钕钇铝石榴石(Nd:YAG)激光黄斑损伤,并进行了为期6年的随访评估。一名23岁女性在未佩戴防护眼镜的情况下意外暴露于调Q Nd:YAG激光下。初次评估时,其患眼的最佳矫正视力为右眼20/100。眼底检查发现黄斑激光灼伤和玻璃体出血。给予60毫克泼尼松龙口服皮质类固醇,每周递减10毫克。暴露19天后,眼底检查发现一个明显的视网膜前膜,该膜在6个月内消退。患眼的最佳矫正视力仍为右眼20/100。该临床过程与先前报道的包括玻璃体出血和随后视网膜前膜形成的病例相似。然而,尽管视网膜前膜自发消退,但视力并未恢复,在6年随访时,既没有脉络膜新生血管形成,也没有黄斑裂孔形成。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/e2366ef110dc/kjo-23-207-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/3b23c285a0de/kjo-23-207-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/b86f264120a8/kjo-23-207-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/2b9fe72df066/kjo-23-207-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/88258dfa2e1f/kjo-23-207-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/e2366ef110dc/kjo-23-207-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/3b23c285a0de/kjo-23-207-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/b86f264120a8/kjo-23-207-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/2b9fe72df066/kjo-23-207-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/88258dfa2e1f/kjo-23-207-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3b8b/2739961/e2366ef110dc/kjo-23-207-g005.jpg

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本文引用的文献

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Retina. 1996;16(1):42-6. doi: 10.1097/00006982-199616010-00008.
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